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Injury in Minipig Parotid Glands following Fractionated Exposure to 30 Gy of Ionizing Radiation.
Wang, Ren-Sheng; Zhang, Ting-Ting; Xu, Zhuo-Hua; Hu, Kai; Shi, Yu; Long, Sha; Yue, Hai-Ying.
Afiliación
  • Wang RS; Department of Radiotherapy, First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi, China 13807806008@163.com.
  • Zhang TT; Department of Radiotherapy, First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi, China.
  • Xu ZH; Department of Radiotherapy, First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi, China.
  • Hu K; Department of Radiotherapy, First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi, China.
  • Shi Y; Department of Radiotherapy, First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi, China.
  • Long S; Department of Radiotherapy, First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi, China.
  • Yue HY; Department of Radiotherapy, First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi, China.
Otolaryngol Head Neck Surg ; 151(1): 100-6, 2014 07.
Article en En | MEDLINE | ID: mdl-24675789
OBJECTIVE: To explore the effects of 30 Gy of (60)Co γ-rays on apoptosis and reactive oxygen species (ROS) levels in minipig parotid cells as a possible mechanism for radiation-induced parotid injury. STUDY DESIGN: Experimental study. SETTING: Department of Radiotherapy, First Affiliated Hospital, Guangxi Medical University, Nanning, China. SUBJECTS AND METHODS: Forty male minipigs were divided into control and irradiated groups. Terminal deoxynucleotidyl transferase deoxyuridine triphosphate nick-end labeling was used for detecting apoptosis in the parotid cells, immunohistochemistry, and western blots were used to test expression of the B-cell lymphoma 2 (Bcl-2) and BCL2-associated X (Bax) proteins, and reverse transcription polymerase chain reaction was used to analyze the expression of Bcl-2, Bax, p53, and caspase-3 messenger ribonucleic acid. An enzyme-linked immunosorbent assay was used to detect ROS levels in the parotid tissue. RESULTS: At each time point, the apoptotic rates in the irradiated group were higher than those in the control group. Furthermore, the ROS and expression levels of Bax, p53, and caspase-3 messenger ribonucleic acid and proteins gradually increased and were higher than those in the control group. Conversely, the expression of Bcl-2 was decreased in the irradiated group (P < .05). CONCLUSIONS: Ionizing radiation induces the production of ROS and promotes changes in the expression of several apoptotic proteins, which increases apoptosis and likely contributes to the mechanism of radiation-induced parotid injury.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Glándula Parótida / Especies Reactivas de Oxígeno / Apoptosis / Proteína X Asociada a bcl-2 / Rayos gamma Límite: Animals Idioma: En Revista: Otolaryngol Head Neck Surg Asunto de la revista: OTORRINOLARINGOLOGIA Año: 2014 Tipo del documento: Article País de afiliación: China Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Glándula Parótida / Especies Reactivas de Oxígeno / Apoptosis / Proteína X Asociada a bcl-2 / Rayos gamma Límite: Animals Idioma: En Revista: Otolaryngol Head Neck Surg Asunto de la revista: OTORRINOLARINGOLOGIA Año: 2014 Tipo del documento: Article País de afiliación: China Pais de publicación: Reino Unido