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Activating FLT3 mutants show distinct gain-of-function phenotypes in vitro and a characteristic signaling pathway profile associated with prognosis in acute myeloid leukemia.
Janke, Hanna; Pastore, Friederike; Schumacher, Daniela; Herold, Tobias; Hopfner, Karl-Peter; Schneider, Stephanie; Berdel, Wolfgang E; Büchner, Thomas; Woermann, Bernhard J; Subklewe, Marion; Bohlander, Stefan K; Hiddemann, Wolfgang; Spiekermann, Karsten; Polzer, Harald.
Afiliación
  • Janke H; Department of Internal Medicine III, University Hospital Munich, Ludwig-Maximilians-University Munich, Germany; Clinical Cooperative Group Leukemia, Helmholtz Center Munich, Germany.
  • Pastore F; Department of Internal Medicine III, University Hospital Munich, Ludwig-Maximilians-University Munich, Germany; Clinical Cooperative Group Leukemia, Helmholtz Center Munich, Germany; German Cancer Consortium (DKTK), Heidelberg, Germany; German Cancer Research Center (DKFZ), Heidelberg, Germany.
  • Schumacher D; Department of Internal Medicine III, University Hospital Munich, Ludwig-Maximilians-University Munich, Germany.
  • Herold T; Department of Internal Medicine III, University Hospital Munich, Ludwig-Maximilians-University Munich, Germany; Clinical Cooperative Group Leukemia, Helmholtz Center Munich, Germany.
  • Hopfner KP; Department of Biochemistry, Gene Center, Ludwig-Maximilians-University Munich, Germany.
  • Schneider S; Department of Internal Medicine III, University Hospital Munich, Ludwig-Maximilians-University Munich, Germany.
  • Berdel WE; Department of Medicine A, Hematology, Oncology and Pneumology, University Muenster, Germany.
  • Büchner T; Department of Medicine A, Hematology, Oncology and Pneumology, University Muenster, Germany.
  • Woermann BJ; German Society of Hematology and Oncology, Berlin, Germany.
  • Subklewe M; Department of Internal Medicine III, University Hospital Munich, Ludwig-Maximilians-University Munich, Germany; German Cancer Consortium (DKTK), Heidelberg, Germany; German Cancer Research Center (DKFZ), Heidelberg, Germany; Clinical Cooperative Group Immunotherapy, Helmholtz Center Munich, Germany.
  • Bohlander SK; Department of Molecular Medicine and Pathology, University of Auckland, New Zealand.
  • Hiddemann W; Department of Internal Medicine III, University Hospital Munich, Ludwig-Maximilians-University Munich, Germany; Clinical Cooperative Group Leukemia, Helmholtz Center Munich, Germany; German Cancer Consortium (DKTK), Heidelberg, Germany; German Cancer Research Center (DKFZ), Heidelberg, Germany.
  • Spiekermann K; Department of Internal Medicine III, University Hospital Munich, Ludwig-Maximilians-University Munich, Germany; Clinical Cooperative Group Leukemia, Helmholtz Center Munich, Germany; German Cancer Consortium (DKTK), Heidelberg, Germany; German Cancer Research Center (DKFZ), Heidelberg, Germany.
  • Polzer H; Department of Internal Medicine III, University Hospital Munich, Ludwig-Maximilians-University Munich, Germany; Clinical Cooperative Group Leukemia, Helmholtz Center Munich, Germany; German Cancer Consortium (DKTK), Heidelberg, Germany; German Cancer Research Center (DKFZ), Heidelberg, Germany.
PLoS One ; 9(3): e89560, 2014.
Article en En | MEDLINE | ID: mdl-24608088
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Leucemia Mieloide Aguda / Tirosina Quinasa 3 Similar a fms Tipo de estudio: Prognostic_studies / Risk_factors_studies Límite: Animals / Humans Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2014 Tipo del documento: Article País de afiliación: Alemania Pais de publicación: Estados Unidos
Texto completo
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Leucemia Mieloide Aguda / Tirosina Quinasa 3 Similar a fms Tipo de estudio: Prognostic_studies / Risk_factors_studies Límite: Animals / Humans Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2014 Tipo del documento: Article País de afiliación: Alemania Pais de publicación: Estados Unidos