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Urocortin 2 treatment is protective in excitotoxic retinal degeneration.
Szabadfi, K; Kiss, P; Reglodi, D; Fekete, E M; Tamas, A; Danyadi, B; Atlasz, T; Gabriel, R.
Afiliación
  • Szabadfi K; University of Pécs Department of Experimental Zoology and Neurobiology, Faculty of Sciences Ifjúság útja 6 H-7624 Pécs Hungary.
  • Kiss P; PTE-MTA "Lendület" PACAP Research Team Department of Anatomy Pécs Hungary.
  • Reglodi D; PTE-MTA "Lendület" PACAP Research Team Department of Anatomy Pécs Hungary.
  • Fekete EM; The Scripps Research Institute La Jolla CA USA University of Wisconsin-Madison Department of Psychiatry Madison WI USA.
  • Tamas A; PTE-MTA "Lendület" PACAP Research Team Department of Anatomy Pécs Hungary.
  • Danyadi B; PTE-MTA "Lendület" PACAP Research Team Department of Anatomy Pécs Hungary.
  • Atlasz T; University of Pécs Department of Sportbiology Pécs Hungary.
  • Gabriel R; University of Pécs Department of Experimental Zoology and Neurobiology, Faculty of Sciences Ifjúság útja 6 H-7624 Pécs Hungary.
Acta Physiol Hung ; 101(1): 67-76, 2014 Mar.
Article en En | MEDLINE | ID: mdl-24311224
Urocortin 2 (Ucn 2) is a corticotrop releasing factor paralog peptide with many physiological functions and it has widespread distribution. There are some data on the cytoprotective effects of Ucn 2, but less is known about its neuro- and retinoprotective actions. We have previously shown that Ucn 2 is protective in ischemia-induced retinal degeneration. The aim of the present study was to examine the protective potential of Ucn 2 in monosodium-glutamate (MSG)-induced retinal degeneration by routine histology and to investigate cell-type specific effects by immunohistochemistry. Rat pups received MSG applied on postnatal days 1, 5 and 9 and Ucn 2 was injected intravitreally into one eye. Retinas were processed for histology and immunocytochemistry after 3 weeks. Immunolabeling was determined for glial fibrillary acidic protein, vesicular glutamate transporter 1, protein kinase Cα, calbindin, parvalbumin and calretinin. Retinal tissue from animals treated with MSG showed severe degeneration compared to normal retinas, but intravitreal Ucn 2 treatment resulted in a retained retinal structure both at histological and neurochemical levels: distinct inner retinal layers and rescued inner retinal cells (different types of amacrine and rod bipolar cells) could be observed. These findings support the neuroprotective function of Ucn 2 in MSG-induced retinal degeneration.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Retina / Degeneración Retiniana / Glutamato de Sodio / Fármacos Neuroprotectores / Urocortinas Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Acta Physiol Hung Año: 2014 Tipo del documento: Article Pais de publicación: Hungria

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Retina / Degeneración Retiniana / Glutamato de Sodio / Fármacos Neuroprotectores / Urocortinas Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Acta Physiol Hung Año: 2014 Tipo del documento: Article Pais de publicación: Hungria