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[Involvement of matrix metalloproteinases in acute coronary syndrome (ACS)]. / Participación de las metaloproteinasas de matriz en el síndrome isquémico coronario agudo (SICA).
Pérez-Hernández, Nonanzit; Ibanes-Gutiérrez, Cyntia; Vargas-Alarcón, Gilberto; Martínez-Rodríguez, Nancy; Monroy-Muñoz, Irma Eloísa; Valente-Acosta, Benjamín; Pérez-Méndez, Oscar; Barrera-Ramírez, Roxana; Juárez-Cedillo, Teresa; Rodríguez Pérez, José Manuel.
Afiliación
  • Pérez-Hernández N; Departamento de Biología Molecular, Instituto Nacional de Cardiología Ignacio Chávez, México, D.F. josemanuel_rodriguezperez@yahoo.com.mx.
Gac Med Mex ; 149(6): 655-67, 2013.
Article en Es | MEDLINE | ID: mdl-24276189
Matrix metalloproteinases (MMP) are enzymes that degrade extracellular matrix (ECM) proteins and regulate both their accumulation and composition. The MMP are involved in the atherosclerotic process since they contribute to the formation of the plaque and its subsequent rupture. This last step triggers the myocardial ischemia that will be clinically reflected as an acute coronary syndrome (ACS). Thus, MMP activity is a key to whether ACS develops or not. With an elevated transcription rate of the genes that codify these proteinases comes a higher enzymatic activity. This explains that if a polymorphism in the mentioned genes modifies transcription, there could be a predisposition to developing ACS. Several studies reveal that certain genetic variations in MMP-1, -2, -3, -7, -8, -9, -12, and -14 have an important role either as risk factors or as protective factors for the expression of ACS.
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Metaloproteinasas de la Matriz / Síndrome Coronario Agudo Tipo de estudio: Risk_factors_studies Límite: Humans Idioma: Es Revista: Gac Med Mex Año: 2013 Tipo del documento: Article Pais de publicación: México
Buscar en Google
Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Metaloproteinasas de la Matriz / Síndrome Coronario Agudo Tipo de estudio: Risk_factors_studies Límite: Humans Idioma: Es Revista: Gac Med Mex Año: 2013 Tipo del documento: Article Pais de publicación: México