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The class I-specific HDAC inhibitor MS-275 modulates the differentiation potential of mouse embryonic stem cells.
Franci, Gianluigi; Casalino, Laura; Petraglia, Francesca; Miceli, Marco; Menafra, Roberta; Radic, Branka; Tarallo, Valeria; Vitale, Monica; Scarfò, Marzia; Pocsfalvi, Gabriella; Baldi, Alfonso; Ambrosino, Concetta; Zambrano, Nicola; Patriarca, Eduardo; De Falco, Sandro; Minchiotti, Gabriella; Stunnenberg, Hendrik G; Altucci, Lucia.
Afiliación
  • Franci G; Dipartimento di Biochimica, Biofisica e Patologia Generale, Seconda Università degli Studi di Napoli , Vico L. De Crecchio 7, 80138 Napoli , Italy ; Department of Molecular Biology, Faculties of Science and Medicine, Radboud University, Nijmegen Center for Molecular Life Sciences , 6500 HB Nijmegen , The Netherlands.
Biol Open ; 2(10): 1070-7, 2013.
Article en En | MEDLINE | ID: mdl-24167717
Exploitation of embryonic stem cells (ESC) for therapeutic use and biomedical applications is severely hampered by the risk of teratocarcinoma formation. Here, we performed a screen of selected epi-modulating compounds and demonstrate that a transient exposure of mouse ESC to MS-275 (Entinostat), a class I histone deacetylase inhibitor (HDAC), modulates differentiation and prevents teratocarcinoma formation. Morphological and molecular data indicate that MS-275-primed ESCs are committed towards neural differentiation, which is supported by transcriptome analyses. Interestingly, in vitro withdrawal of MS-275 reverses the primed cells to the pluripotent state. In vivo, MS275-primed ES cells injected into recipient mice give only rise to benign teratomas but not teratocarcinomas with prevalence of neural-derived structures. In agreement, MS-275-primed ESC are unable to colonize blastocysts. These findings provide evidence that a transient alteration of acetylation alters the ESC fate.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Tipo de estudio: Risk_factors_studies Idioma: En Revista: Biol Open Año: 2013 Tipo del documento: Article País de afiliación: Países Bajos Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Tipo de estudio: Risk_factors_studies Idioma: En Revista: Biol Open Año: 2013 Tipo del documento: Article País de afiliación: Países Bajos Pais de publicación: Reino Unido