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Evaluation of acetylcholinesterase and adenosine deaminase activities in brain and erythrocytes and proinflammatory cytokine levels in rats submitted to neonatal hypoxia-ischemia model.
Pimentel, Victor Camera; Gomes, Jéssica Lopes; Zanini, Daniela; Abdalla, Fátima Husein; da Costa, Pauline; Gonçalves, Jamile Fabbrin; Duarte, Marta Maria Medeiros Frescura; Moretto, Maria Beatriz; Morsch, Vera Maria; Schetinger, Maria Rosa Chitolina.
Afiliación
  • Pimentel VC; Departamento de Química, Centro de Ciências Naturais e Exatas, Universidade Federal de Santa Maria, Campus Universitário, Camobi, Santa Maria, RS, Brazil. victor.capi@yahoo.com.br
Mol Cell Biochem ; 378(1-2): 247-55, 2013 Jun.
Article en En | MEDLINE | ID: mdl-23516038
Perinatal hypoxic-ischemic (HI) brain injury is a common problem with severe neurologic sequelae. The definitive brain injury is a consequence of pathophysiological mechanisms that begin at the moment of HI insult and may extend for days or weeks. In this context, the inflammatory response and the formation of reactive oxygen species seem to play a key role during evolution of brain damage after injury. Thus, the aim of this study was to describe the chronological sequence of acetylcholinesterase (AChE) activity and the lipid peroxidation changes in the cerebral cortex using the classic model of neonatal HI. Furthermore, the erythrocyte AChE and adenosine deaminase (ADA) activities as well as the serum levels of proinflammatory cytokines were assessed. We observed that neonatal HI caused an increase of lipid peroxidation immediately after HI insult, which remained for several days afterward. There was a time-related change in the AChE activity in the cerebral cortex and the same was observed in erythrocyte AChE and ADA activities. In addition, immediately after HI, ADA activity showed a strong positive correlation with all proinflammatory cytokines assessed. Together, these findings may help the understanding of some mechanism related to the pathophysiology of neonatal HI, therefore highlighting the putative therapeutic targets to minimize brain injury and enhance recovery.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Acetilcolinesterasa / Adenosina Desaminasa / Corteza Cerebral / Isquemia Encefálica / Citocinas / Eritrocitos Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Mol Cell Biochem Año: 2013 Tipo del documento: Article País de afiliación: Brasil Pais de publicación: Países Bajos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Acetilcolinesterasa / Adenosina Desaminasa / Corteza Cerebral / Isquemia Encefálica / Citocinas / Eritrocitos Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Mol Cell Biochem Año: 2013 Tipo del documento: Article País de afiliación: Brasil Pais de publicación: Países Bajos