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Regulation of matrixmetalloproteinase-3 and matrixmetalloproteinase-13 by SUMO-2/3 through the transcription factor NF-κB.
Frank, Svetlana; Peters, Marvin A; Wehmeyer, Corinna; Strietholt, Simon; Koers-Wunrau, Christina; Bertrand, Jessica; Heitzmann, Marianne; Hillmann, Anja; Sherwood, Joanna; Seyfert, Christine; Gay, Steffen; Pap, Thomas.
Afiliación
  • Frank S; Institute of Experimental Musculoskeletal Medicine, University Hospital Muenster, , Muenster, Germany.
Ann Rheum Dis ; 72(11): 1874-81, 2013 Nov.
Article en En | MEDLINE | ID: mdl-23417988
OBJECTIVE: Based on previous data that have linked the small ubiquitin-like modifier-1 (SUMO-1) to the pathogenesis of rheumatoid arthritis (RA), we have investigated the expression of the highly homologous SUMO family members SUMO-2/3 in human RA and in the human tumour necrosis factor α transgenic (hTNFtg) mouse model of RA and studied their role in regulating disease specific matrixmetalloproteinases (MMPs). METHODS: Synovial tissue was obtained from RA and osteoarthritis (OA) patients and used for histological analyses as well as for the isolation of synovial fibroblasts (SFs). The expression of SUMO-2/3 in RA and OA patients as well as in hTNFtg and wild type mice was studied by PCR, western blot and immunostaining. SUMO-2/3 was knocked down using small interfering RNA in SFs, and TNF-α induced MMP production was determined by ELISA. Activation of nuclear factor-κB (NF-κB) was determined by a luciferase activity assay and a transcription factor assay in the presence of the NF-κB inhibitor BAY 11-7082. RESULTS: Expression of SUMO-2 and to a lesser extent of SUMO-3 was higher in RA tissues and RASFs compared with OA controls. Similarly, there was increased expression of SUMO-2 in the synovium and in SFs of hTNFtg mice compared with wild type animals. In vitro, the expression of SUMO-2 but not of SUMO-3 was induced by TNF-α. The knockdown of SUMO-2/3 significantly increased the TNF-α and interleukin (IL)-1ß induced expression of MMP-3 and MMP-13, accompanied by increased NF-κB activity. Induction of MMP-3 and MMP-13 was inhibited by blockade of the NF-κB pathway. TNF-α and IL-1ß mediated MMP-1 expression was not regulated by SUMO-2/3. CONCLUSIONS: Collectively, we show that despite their high homology, SUMO-2/3 are differentially regulated by TNF-α and selectively control TNF-α mediated MMP expression via the NF-κB pathway. Therefore, we hypothesise that SUMO-2 contributes to the specific activation of RASF.
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Artritis Reumatoide / FN-kappa B / Factor de Necrosis Tumoral alfa / Metaloproteinasa 3 de la Matriz / Proteínas Modificadoras Pequeñas Relacionadas con Ubiquitina / Metaloproteinasa 13 de la Matriz / Fibroblastos Tipo de estudio: Prognostic_studies Límite: Animals / Humans Idioma: En Revista: Ann Rheum Dis Año: 2013 Tipo del documento: Article País de afiliación: Alemania Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Artritis Reumatoide / FN-kappa B / Factor de Necrosis Tumoral alfa / Metaloproteinasa 3 de la Matriz / Proteínas Modificadoras Pequeñas Relacionadas con Ubiquitina / Metaloproteinasa 13 de la Matriz / Fibroblastos Tipo de estudio: Prognostic_studies Límite: Animals / Humans Idioma: En Revista: Ann Rheum Dis Año: 2013 Tipo del documento: Article País de afiliación: Alemania Pais de publicación: Reino Unido