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Single nucleotide polymorphism array lesions, TET2, DNMT3A, ASXL1 and CBL mutations are present in systemic mastocytosis.
Traina, Fabiola; Visconte, Valeria; Jankowska, Anna M; Makishima, Hideki; O'Keefe, Christine L; Elson, Paul; Han, Yingchun; Hsieh, Fred H; Sekeres, Mikkael A; Mali, Raghuveer Singh; Kalaycio, Matt; Lichtin, Alan E; Advani, Anjali S; Duong, Hien K; Copelan, Edward; Kapur, Reuben; Olalla Saad, Sara T; Maciejewski, Jaroslaw P; Tiu, Ramon V.
Afiliación
  • Traina F; Department of Translational Hematology and Oncology Research, Taussig Cancer Institute, Cleveland Clinic, Cleveland, Ohio, United States of America.
PLoS One ; 7(8): e43090, 2012.
Article en En | MEDLINE | ID: mdl-22905207
We hypothesized that analysis of single nucleotide polymorphism arrays (SNP-A) and new molecular defects may provide new insight in the pathogenesis of systemic mastocytosis (SM). SNP-A karyotyping was applied to identify recurrent areas of loss of heterozygosity and bidirectional sequencing was performed to evaluate the mutational status of TET2, DNMT3A, ASXL1, EZH2, IDH1/IDH2 and the CBL gene family. Overall survival (OS) was analyzed using the Kaplan-Meier method. We studied a total of 26 patients with SM. In 67% of SM patients, SNP-A karyotyping showed new chromosomal abnormalities including uniparental disomy of 4q and 2p spanning TET2/KIT and DNMT3A. Mutations in TET2, DNMT3A, ASXL1 and CBL were found in 23%, 12%, 12%, and 4% of SM patients, respectively. No mutations were observed in EZH2 and IDH1/IDH2. Significant differences in OS were observed for SM mutated patients grouped based on the presence of combined TET2/DNMT3A/ASXL1 mutations independent of KIT (P = 0.04) and sole TET2 mutations (P<0.001). In conclusion, TET2, DNMT3A and ASXL1 mutations are also present in mastocytosis and these mutations may affect prognosis, as demonstrated by worse OS in mutated patients.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Proteínas Represoras / Regulación de la Expresión Génica / Proteínas Proto-Oncogénicas / Polimorfismo de Nucleótido Simple / Mastocitosis Sistémica / ADN (Citosina-5-)-Metiltransferasas / Proteínas de Unión al ADN / Proteínas Proto-Oncogénicas c-cbl Tipo de estudio: Prognostic_studies Límite: Adolescent / Adult / Aged / Female / Humans / Middle aged Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2012 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Proteínas Represoras / Regulación de la Expresión Génica / Proteínas Proto-Oncogénicas / Polimorfismo de Nucleótido Simple / Mastocitosis Sistémica / ADN (Citosina-5-)-Metiltransferasas / Proteínas de Unión al ADN / Proteínas Proto-Oncogénicas c-cbl Tipo de estudio: Prognostic_studies Límite: Adolescent / Adult / Aged / Female / Humans / Middle aged Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2012 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos