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Laropiprant attenuates EP3 and TP prostanoid receptor-mediated thrombus formation.
Philipose, Sonia; Konya, Viktoria; Lazarevic, Mirjana; Pasterk, Lisa M; Marsche, Gunther; Frank, Sasa; Peskar, Bernhard A; Heinemann, Akos; Schuligoi, Rufina.
Afiliación
  • Philipose S; Institute of Experimental and Clinical Pharmacology, Medical University of Graz, Graz, Austria.
PLoS One ; 7(8): e40222, 2012.
Article en En | MEDLINE | ID: mdl-22870195
The use of the lipid lowering agent niacin is hampered by a frequent flush response which is largely mediated by prostaglandin (PG) D(2). Therefore, concomitant administration of the D-type prostanoid (DP) receptor antagonist laropiprant has been proposed to be a useful approach in preventing niacin-induced flush. However, antagonizing PGD(2), which is a potent inhibitor of platelet aggregation, might pose the risk of atherothrombotic events in cardiovascular disease. In fact, we found that in vitro treatment of platelets with laropiprant prevented the inhibitory effects of PGD(2) on platelet function, i.e. platelet aggregation, Ca(2+) flux, P-selectin expression, activation of glycoprotein IIb/IIIa and thrombus formation. In contrast, laropiprant did not prevent the inhibitory effects of acetylsalicylic acid or niacin on thrombus formation. At higher concentrations, laropiprant by itself attenuated platelet activation induced by thromboxane (TP) and E-type prostanoid (EP)-3 receptor stimulation, as demonstrated in assays of platelet aggregation, Ca(2+) flux, P-selectin expression, and activation of glycoprotein IIb/IIIa. Inhibition of platelet function exerted by EP4 or I-type prostanoid (IP) receptors was not affected by laropiprant. These in vitro data suggest that niacin/laropiprant for the treatment of dyslipidemias might have a beneficial profile with respect to platelet function and thrombotic events in vascular disease.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Coagulación Sanguínea / Plaquetas / Agregación Plaquetaria / Regulación de la Expresión Génica / Subtipo EP3 de Receptores de Prostaglandina E / Indoles Límite: Female / Humans / Male Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2012 Tipo del documento: Article País de afiliación: Austria Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Coagulación Sanguínea / Plaquetas / Agregación Plaquetaria / Regulación de la Expresión Génica / Subtipo EP3 de Receptores de Prostaglandina E / Indoles Límite: Female / Humans / Male Idioma: En Revista: PLoS One Asunto de la revista: CIENCIA / MEDICINA Año: 2012 Tipo del documento: Article País de afiliación: Austria Pais de publicación: Estados Unidos