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Peroxisome proliferator-activated receptor γ-regulated cathepsin D is required for lipid antigen presentation by dendritic cells.
Nakken, Britt; Varga, Tamas; Szatmari, Istvan; Szeles, Lajos; Gyongyosi, Adrienn; Illarionov, Petr A; Dezso, Balazs; Gogolak, Peter; Rajnavolgyi, Eva; Nagy, Laszlo.
Afiliación
  • Nakken B; Department of Biochemistry and Molecular Biology, Research Center for Molecular Medicine, University of Debrecen, Debrecen 4010, Hungary.
J Immunol ; 187(1): 240-7, 2011 Jul 01.
Article en En | MEDLINE | ID: mdl-21632707
It is well established that dendritic cells (DCs) take up, process, and present lipid Ags in complex with CD1d molecules to invariant NKT cells. The lipid-activated transcription factor, peroxisome proliferator-activated receptor γ (PPARγ), has previously been shown to regulate CD1d expression in human monocyte-derived DCs, providing a link between lipid metabolism and lipid Ag presentation. We report that PPARγ regulates the expression of a lysosomal protease, cathepsin D (CatD), in human monocyte-derived DCs. Inhibition of CatD specifically reduced the expansion of invariant NKT cells and furthermore resulted in decreased maturation of saposins, a group of lipid transfer proteins required for lysosomal lipid Ag processing and loading. These results reveal a novel mechanism of lipid Ag presentation and identify CatD as a key component of this machinery and firmly place PPARγ as the transcriptional regulator linking lipid metabolism and lipid Ag processing.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Células Dendríticas / Catepsina D / Presentación de Antígeno / PPAR gamma / Lipoproteínas Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Revista: J Immunol Año: 2011 Tipo del documento: Article País de afiliación: Hungria Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Células Dendríticas / Catepsina D / Presentación de Antígeno / PPAR gamma / Lipoproteínas Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Revista: J Immunol Año: 2011 Tipo del documento: Article País de afiliación: Hungria Pais de publicación: Estados Unidos