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Twenty-five novel mutations including duplications in the ATP7A gene.
Moizard, M-P; Ronce, N; Blesson, S; Bieth, E; Burglen, L; Mignot, C; Mortemousque, I; Marmin, N; Dessay, B; Danesino, C; Feillet, F; Castelnau, P; Toutain, A; Moraine, C; Raynaud, M.
Afiliación
  • Moizard MP; CHRU de Tours, Service de Génétique, Tours, F-37044, France INSERM U930, Tours, F-37044, France CHU Hôpital Purpan, Service de Génétique médicale, Toulouse, F-31059, France CHU Hôpital d'Enfants Armand-Trousseau, AP-HP, Service de Génétique et Embryologie médicales, Paris, F-75571, France CHU Hôpital d'Enfants Armand-Trousseau, AP-HP, Service de Neuropédiatrie, Paris, F-75012, France Genetica Medica, Università di Pavia, Fondazione IRCCS S. Matteo, Pavia, I-27100, Italie Centre de Référence des
Clin Genet ; 79(3): 243-53, 2011 Mar.
Article en En | MEDLINE | ID: mdl-21208200
Twenty-five novel mutations including duplications in the ATP7A gene. Menkes disease (MD) and occipital horn syndrome (OHS) are allelic X-linked recessive copper deficiency disorders resulting from ATP7A gene mutations. MD is a severe condition leading to progressive neurological degeneration and death in early childhood, whereas OHS has a milder phenotype with mainly connective tissue abnormalities. Until now, molecular analyses have revealed only deletions and point mutations in both diseases. This study reports new molecular data in a series of 40 patients referred for either MD or OHS. We describe 23 point mutations (9 missense mutations, 7 splice site variants, 4 nonsense mutations, and 3 small insertions or deletions) and 7 intragenic deletions. Of these, 18 point mutations and 3 deletions are novel. Furthermore, our finding of four whole exon duplications enlarges the mutation spectrum in the ATP7A gene. ATP7A alterations were found in 85% of cases. Of these alterations, two thirds were point mutations and the remaining one third consisted of large rearrangements. We found that 66.6% of point mutations resulted in impaired ATP7A transcript splicing, a phenomenon more frequent than expected. This finding enabled us to confirm the pathogenic role of ATP7A mutations, particularly in missense and splice site variants.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Eliminación de Secuencia / Mutación Puntual / Adenosina Trifosfatasas / Duplicación de Gen / Cutis Laxo / Proteínas de Transporte de Catión / Síndrome de Ehlers-Danlos / Síndrome del Pelo Ensortijado Límite: Female / Humans / Male Idioma: En Revista: Clin Genet Año: 2011 Tipo del documento: Article Pais de publicación: Dinamarca

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Eliminación de Secuencia / Mutación Puntual / Adenosina Trifosfatasas / Duplicación de Gen / Cutis Laxo / Proteínas de Transporte de Catión / Síndrome de Ehlers-Danlos / Síndrome del Pelo Ensortijado Límite: Female / Humans / Male Idioma: En Revista: Clin Genet Año: 2011 Tipo del documento: Article Pais de publicación: Dinamarca