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Deep brain stimulation can suppress pathological synchronisation in parkinsonian patients.
Eusebio, A; Thevathasan, W; Doyle Gaynor, L; Pogosyan, A; Bye, E; Foltynie, T; Zrinzo, L; Ashkan, K; Aziz, T; Brown, P.
Afiliación
  • Eusebio A; Sobell Department of Motor Neuroscience and Movement Disorders, Institute of Neurology, London, UK.
J Neurol Neurosurg Psychiatry ; 82(5): 569-73, 2011 May.
Article en En | MEDLINE | ID: mdl-20935326
BACKGROUND: Although deep brain stimulation (DBS) of the subthalamic nucleus (STN) is a highly effective therapeutic intervention in severe Parkinson's disease, its mechanism of action remains unclear. One possibility is that DBS suppresses local pathologically synchronised oscillatory activity. METHODS: To explore this, the authors recorded from DBS electrodes implanted in the STN of 16 patients with Parkinson's disease during simultaneous stimulation (pulse width 60 µs; frequency 130 Hz) of the same target using a specially designed amplifier. The authors analysed data from 25 sides. RESULTS: The authors found that DBS progressively suppressed peaks in local field potential activity at frequencies between 11 and 30 Hz as voltage was increased beyond a stimulation threshold of 1.5 V. Median peak power had fallen to 54% of baseline values by a stimulation intensity of 3.0 V. CONCLUSION: The findings suggest that DBS can suppress pathological 11-30 Hz activity in the vicinity of stimulation in patients with Parkinson's disease. This suppression occurs at stimulation voltages that are clinically effective.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Enfermedad de Parkinson / Encéfalo / Sincronización Cortical / Estimulación Encefálica Profunda Límite: Humans Idioma: En Revista: J Neurol Neurosurg Psychiatry Año: 2011 Tipo del documento: Article Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Enfermedad de Parkinson / Encéfalo / Sincronización Cortical / Estimulación Encefálica Profunda Límite: Humans Idioma: En Revista: J Neurol Neurosurg Psychiatry Año: 2011 Tipo del documento: Article Pais de publicación: Reino Unido