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ZBP-89 enhances Bak expression and causes apoptosis in hepatocellular carcinoma cells.
To, Ann K Y; Chen, George G; Chan, Ursula P F; Ye, Caiguo; Yun, Jing P; Ho, Rocky L K; Tessier, Art; Merchant, Juanita L; Lai, Paul B S.
Afiliación
  • To AK; Department of Surgery, The Chinese University of Hong Kong, Prince of Wales Hospital, Shatin, New Territories, Hong Kong.
Biochim Biophys Acta ; 1813(1): 222-30, 2011 Jan.
Article en En | MEDLINE | ID: mdl-20850481
ZBP-89 can enhance tumor cells to death stimuli. However, the molecular mechanism leading to the inhibitory effect of ZBP-89 is unknown. In this study, 4 liver cell lines were used to screen for the target of ZBP-89 on cell death pathway. The identified Bak was further analyzed for its role in ZBP-89-mediated apoptosis. The result showed that ZBP-89 significantly and time-dependently induced apoptosis. It significantly upregulated the level of pro-apoptotic Bak. ZBP-89 targeted a region between -457 and -407 of human Bak promoter to stimulate Bak expression based on the findings of Bak promoter luciferase report gene assay and electrophoretic mobility shift assay. ZBP-89-induced Bak increase and ZBP-89-mediated apoptosis were markedly suppressed by Bak siRNA, confirming that Bak was specifically targeted by ZBP-89 to facilitate apoptosis. In conclusion, this study demonstrated that ZBP-89 significantly induced apoptosis of HCC cells via promoting Bak level.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Factores de Transcripción / Apoptosis / Carcinoma Hepatocelular / Proteínas de Unión al ADN / Proteína Destructora del Antagonista Homólogo bcl-2 / Neoplasias Hepáticas Tipo de estudio: Etiology_studies / Prognostic_studies Límite: Humans Idioma: En Revista: Biochim Biophys Acta Año: 2011 Tipo del documento: Article País de afiliación: Hong Kong Pais de publicación: Países Bajos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Factores de Transcripción / Apoptosis / Carcinoma Hepatocelular / Proteínas de Unión al ADN / Proteína Destructora del Antagonista Homólogo bcl-2 / Neoplasias Hepáticas Tipo de estudio: Etiology_studies / Prognostic_studies Límite: Humans Idioma: En Revista: Biochim Biophys Acta Año: 2011 Tipo del documento: Article País de afiliación: Hong Kong Pais de publicación: Países Bajos