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Direct, help-independent priming of CD8+ T cells by adeno-associated virus-transduced hepatocytes.
Wuensch, Sherry A; Spahn, Jessica; Crispe, Ian N.
Afiliación
  • Wuensch SA; The David H. Smith Center for Vaccine Biology and Immunology, University of Rochester Medical Center, Rochester, NY, USA.
Hepatology ; 52(3): 1068-77, 2010 Sep.
Article en En | MEDLINE | ID: mdl-20607836
UNLABELLED: Both hepatitis B and C viruses frequently establish chronic infection, raising the question whether T cells are poorly primed in the liver. To determine the role of different cell types in the activation of CD8+ T cells against hepatocellular antigens, we used an Adeno-associated virus to deliver ovalbumin to hepatocytes. In contrast to CD8+ T cells, CD4+ T cells were not activated. The CD8+ T cells were activated even in the absence of endogenous CD4+ T cells; however, in the liver, these cells were high in the programmed death-1 protein and low in CD127. Chimera experiments revealed that these CD8+ T cells were activated on a solid tissue cell. CONCLUSION: Priming of CD8+ T cells directly on nonhematopoietic cells, in the absence of CD4+ T cell help, results in suboptimal T cell activation. This could explain the impaired function of CD8+ T cells seen in chronic liver infection.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Adenoviridae / Linfocitos T CD8-positivos / Hepatocitos / Hígado Tipo de estudio: Risk_factors_studies Límite: Animals Idioma: En Revista: Hepatology Año: 2010 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Adenoviridae / Linfocitos T CD8-positivos / Hepatocitos / Hígado Tipo de estudio: Risk_factors_studies Límite: Animals Idioma: En Revista: Hepatology Año: 2010 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos