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Founder mutations in hypertrophic cardiomyopathy patients in the Netherlands.
Christiaans, I; Nannenberg, E A; Dooijes, D; Jongbloed, R J E; Michels, M; Postema, P G; Majoor-Krakauer, D; van den Wijngaard, A; Mannens, M M A M; van Tintelen, J P; van Langen, I M; Wilde, A A M.
Afiliación
  • Christiaans I; Department of Clinical Genetics, Academic Medical Centre, Amsterdam, the Netherlands These authors contributed equally.
Neth Heart J ; 18(5): 248-54, 2010 May.
Article en En | MEDLINE | ID: mdl-20505798
In this part of a series on cardiogenetic founder mutations in the Netherlands, we review the Dutch founder mutations in hypertrophic cardiomyopathy (HCM) patients. HCM is a common autosomal dominant genetic disease affecting at least one in 500 persons in the general population. Worldwide, most mutations in HCM patients are identified in genes encoding sarcomeric proteins, mainly in the myosin-binding protein C gene (MYBPC3, OMIM #600958) and the beta myosin heavy chain gene (MYH7, OMIM #160760). In the Netherlands, the great majority of mutations occur in the MYBPC3, involving mainly three Dutch founder mutations in the MYBPC3 gene, the c.2373_2374insG, the c.2864_2865delCT and the c.2827C>T mutation. In this review, we describe the genetics of HCM, the genotype-phenotype relation of Dutch founder MYBPC3 gene mutations, the prevalence and the geographic distribution of the Dutch founder mutations, and the consequences for genetic counselling and testing. (Neth Heart J 2010;18:248-54.).
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Tipo de estudio: Risk_factors_studies Idioma: En Revista: Neth Heart J Año: 2010 Tipo del documento: Article Pais de publicación: Países Bajos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Tipo de estudio: Risk_factors_studies Idioma: En Revista: Neth Heart J Año: 2010 Tipo del documento: Article Pais de publicación: Países Bajos