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Phenotype, compartmental organization and differential vulnerability of nigral dopaminergic neurons.
González-Hernández, Tomás; Afonso-Oramas, Domingo; Cruz-Muros, Ignacio.
Afiliación
  • González-Hernández T; Department of Anatomy, Faculty of Medicine, University of La Laguna, 38071, La Laguna, Tenerife, Spain. tgonhern@ull.es
J Neural Transm Suppl ; (73): 21-37, 2009.
Article en En | MEDLINE | ID: mdl-20411765
The degeneration of nigral dopaminergic (DA-) neurons is the histopathologic hallmark of Parkinson's disease (PD), but not all nigral DA-cells show the same susceptibility to degeneration. This starts in DA-cells in the ventrolateral and caudal regions of the susbtantia nigra (SN) and progresses to DA-cells in the dorsomedial and rostral regions of the SN and the ventral tegmental area, where many neurons remain intact until the final stages of the disease. This fact indicates a relationship between the topographic distribution of midbrain DA-cells and their differential vulnerability, and the possibility that this differential vulnerability is associated with phenotypic differences between different subpopulations of nigral DA-cells. Studies carried out during the last two decades have contributed to establishing the existence of different compartments of nigral DA-cells according to their neurochemical profile, and a possible relationship between the expression of some factors and the relative vulnerability or resistance of DA-cell subpopulations to degeneration. These aspects are reviewed and discussed here.
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Fenotipo / Sustancia Negra / Dopamina / Regulación de la Expresión Génica / Neuronas Límite: Animals / Humans Idioma: En Revista: J Neural Transm Suppl Año: 2009 Tipo del documento: Article País de afiliación: España Pais de publicación: Austria
Buscar en Google
Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Fenotipo / Sustancia Negra / Dopamina / Regulación de la Expresión Génica / Neuronas Límite: Animals / Humans Idioma: En Revista: J Neural Transm Suppl Año: 2009 Tipo del documento: Article País de afiliación: España Pais de publicación: Austria