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Pore architecture of the ORAI1 store-operated calcium channel.
Zhou, Yubin; Ramachandran, Sweta; Oh-Hora, Masatsugu; Rao, Anjana; Hogan, Patrick G.
Afiliación
  • Zhou Y; Immune Disease Institute and Program in Cellular and Molecular Medicine, Children's Hospital Boston, 200 Longwood Avenue, Boston, MA 02115, USA.
Proc Natl Acad Sci U S A ; 107(11): 4896-901, 2010 Mar 16.
Article en En | MEDLINE | ID: mdl-20194792
ORAI1 is the pore-forming subunit of the calcium release-activated calcium (CRAC) channel, a store-operated channel that is central to Ca(2+) signaling in mammalian cells. Electrophysiological data have shown that the acidic residues E106 in transmembrane helix 1 (TM1) and E190 in TM3 contribute to the high selectivity of ORAI1 channels for Ca(2+). We have examined the pore architecture of the ORAI1 channel using ORAI1 proteins engineered to contain either one or two cysteine residues. Disulfide cross-linking shows that ORAI1 assembles as a tetramer or a higher oligomer with TM1 centrally located. Cysteine side chains projecting from TM1 at position 88, 95, 102, or 106 cross-link efficiently to the corresponding side chain in a second ORAI1 monomer. Cysteine residues at position 190 or at surrounding positions in TM3 do not cross-link. We conclude that E106 residues in wild-type ORAI1 are positioned to form a Ca(2+) binding site in the channel pore and that E190 interacts less directly with ions traversing the pore. The cross-linking data further identify a relatively rigid segment of TM1 adjacent to E106 that is likely to contribute to the selectivity filter.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Canales de Calcio / Membrana Celular Límite: Animals / Humans Idioma: En Revista: Proc Natl Acad Sci U S A Año: 2010 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Canales de Calcio / Membrana Celular Límite: Animals / Humans Idioma: En Revista: Proc Natl Acad Sci U S A Año: 2010 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos