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Targeted inhibition of the serotonin 5HT2A receptor improves coronary patency in an in vivo model of recurrent thrombosis.
Przyklenk, K; Frelinger, A L; Linden, M D; Whittaker, P; Li, Y; Barnard, M R; Adams, J; Morgan, M; Al-Shamma, H; Michelson, A D.
Afiliación
  • Przyklenk K; Center for Platelet Function Studies, Department of Emergency Medicine, University of Massachusetts Medical School, Worcester, MA, USA. kprzykle@med.wayne.edu
J Thromb Haemost ; 8(2): 331-40, 2010 Feb.
Article en En | MEDLINE | ID: mdl-19922435
BACKGROUND: Release of serotonin and activation of serotonin 5HT2A receptors on platelet surfaces is a potent augmentative stimulus for platelet aggregation. However, earlier-generation serotonin receptor antagonists were not successfully exploited as antiplatelet agents, possibly owing to their lack of specificity for the 5HT2A receptor subtype. OBJECTIVE: To assess whether targeted inhibition of the serotonin 5HT2A receptor attenuates recurrent thrombosis and improves coronary patency in an in vivo canine model mimicking unstable angina. METHODS: In protocol 1, anesthetized dogs were pretreated with a novel, selective inverse agonist of the 5HT2A receptor (APD791) or saline. Recurrent coronary thrombosis was then initiated by coronary artery injury+stenosis, and coronary patency was monitored for 3 h. Protocol 2 was similar, except that: (i) treatment with APD791 or saline was begun 1 h after the onset of recurrent thrombosis; (ii) template bleeding time was measured; and (iii) blood samples were obtained for in vitro flow cytometric assessment of platelet responsiveness to serotonin. RESULTS: APD791 attenuated recurrent thrombosis, irrespective of the time of treatment: in both protocols, flow-time area (index of coronary patency; normalized to baseline coronary flow) averaged 58-59% (P<0.01) following administration of APD791 vs. 21-28% in saline controls. Moreover, the in vivo antithrombotic effect of APD791 was not accompanied by increased bleeding, but was associated with significant and selective inhibition of serotonin-mediated platelet activation. CONCLUSION: 5HT2A receptor inhibition with APD791, even when initiated after the onset of recurrent thrombosis, improves coronary patency in the in vivo canine model.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Pirazoles / Antagonistas de la Serotonina / Benzamidas / Plaquetas / Trombosis Coronaria / Grado de Desobstrucción Vascular / Inhibidores de Agregación Plaquetaria / Morfolinas / Antagonistas del Receptor de Serotonina 5-HT2 / Fibrinolíticos Tipo de estudio: Guideline / Prognostic_studies Límite: Animals Idioma: En Revista: J Thromb Haemost Asunto de la revista: HEMATOLOGIA Año: 2010 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Pirazoles / Antagonistas de la Serotonina / Benzamidas / Plaquetas / Trombosis Coronaria / Grado de Desobstrucción Vascular / Inhibidores de Agregación Plaquetaria / Morfolinas / Antagonistas del Receptor de Serotonina 5-HT2 / Fibrinolíticos Tipo de estudio: Guideline / Prognostic_studies Límite: Animals Idioma: En Revista: J Thromb Haemost Asunto de la revista: HEMATOLOGIA Año: 2010 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido