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Neuroplasticity and neuroprotection in enteric neurons: role of epithelial cells.
Moriez, Raphaël; Abdo, Hind; Chaumette, Tanguy; Faure, Magali; Lardeux, Bernard; Neunlist, Michel.
Afiliación
  • Moriez R; INSERM, U913, 1, Place Alexis Ricordeau - 3HNB, Nantes F-44093, France.
Biochem Biophys Res Commun ; 382(3): 577-82, 2009 May 08.
Article en En | MEDLINE | ID: mdl-19302981
Neurons of enteric nervous system (ENS) regulate intestinal epithelial cells (IEC) functions but whether IEC can impact upon the neurochemical coding and survival of enteric neurons remain unknown. Neuro-epithelial interactions were studied using a coculture model composed of IEC lines and primary culture of rat ENS or human neuroblastoma cells (SH-SY5Y). Neurochemical coding of enteric neurons was analysed by immunohistochemistry and quantitative PCR. Neuroprotective effects of IEC were tested by measuring neuron specific enolase (NSE) release or cell permeability to 7-amino-actinomycin D (7-AAD). Following coculture with IEC, the percentage of VIP-immunoreactive (IR) neurons but not NOS-IR and VIP mRNA expression were significantly increased. IEC significantly reduced dopamine-induced NSE release and 7-AAD permeability in culture of ENS and SH-SY5Y, respectively. Finally, we showed that NGF had neuroprotective effects but reduced VIP expression in enteric neurons. In conclusion, our study identified a novel role for IEC in the regulation of enteric neuronal properties.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Mucosa Intestinal / Intestinos / Plasticidad Neuronal / Neuronas Límite: Animals / Humans Idioma: En Revista: Biochem Biophys Res Commun Año: 2009 Tipo del documento: Article País de afiliación: Francia Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Mucosa Intestinal / Intestinos / Plasticidad Neuronal / Neuronas Límite: Animals / Humans Idioma: En Revista: Biochem Biophys Res Commun Año: 2009 Tipo del documento: Article País de afiliación: Francia Pais de publicación: Estados Unidos