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Promoter polymorphisms modulating HSPA5 expression may increase susceptibility to Taiwanese Alzheimer's disease.
Hsu, W-C; Wang, H-K; Lee, L-C; Fung, H-C; Lin, J-C; Hsu, H-P; Wu, Y-R; Ro, L-S; Hu, F-J; Chang, Y-T; Lee-Chen, G-J; Chen, C-M.
Afiliación
  • Hsu WC; Department of Neurology, Chang Gung Memorial Hospital, Chang Gung University College of Medicine, Taipei, 10507, Taiwan.
J Neural Transm (Vienna) ; 115(11): 1537-43, 2008 Nov.
Article en En | MEDLINE | ID: mdl-18781276
Endoplasmic reticulum (ER) chaperone heat shock 70 kDa protein 5 (HSPA5/GRP78) is known to be involved in the metabolism of amyloid precursor protein and neuronal death in Alzheimer's disease (AD) could arise from dysfunction of the ER. Through a case-control study and an expression assay, we investigated the association of HSPA5 -415 G/A (rs391957), -370 C/T (rs17840761) and -180 del/G (rs3216733) polymorphisms with Taiwanese AD. The overall genotype and allele frequency distribution at the completely linked -415 G/A and -180 del/G sites showed significant difference between AD cases and controls (P = 0.020 and 0.009, respectively). A decrease in risk of developing AD was demonstrated for -415 AA/-180 GG genotype [OR = 0.38, 95% confidence interval (CI) = 0.18-0.75, P = 0.007] and -415 A/-180 G allele (OR = 0.69, 95% CI = 0.51-0.91, P = 0.009). The HSPA5 transcriptional activity of the -415 A/-180 G allele was significantly lower than that of the -415 G/-180 del alleles, whereas induction of HSPA5 expression after ER stress was markedly increased in the cells with the -415 A/-180 G allele. Therefore, our preliminary results may suggest a protective role of the HSPA5 -415 A/-180 G allele in Taiwanese AD susceptibility.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Chaperonas Moleculares / Enfermedad de Alzheimer / Proteínas de Choque Térmico Tipo de estudio: Etiology_studies / Observational_studies / Risk_factors_studies Límite: Aged / Female / Humans / Male País/Región como asunto: Asia Idioma: En Revista: J Neural Transm (Vienna) Año: 2008 Tipo del documento: Article País de afiliación: Taiwán Pais de publicación: Austria

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Chaperonas Moleculares / Enfermedad de Alzheimer / Proteínas de Choque Térmico Tipo de estudio: Etiology_studies / Observational_studies / Risk_factors_studies Límite: Aged / Female / Humans / Male País/Región como asunto: Asia Idioma: En Revista: J Neural Transm (Vienna) Año: 2008 Tipo del documento: Article País de afiliación: Taiwán Pais de publicación: Austria