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Evaluation of beta-cell mass and function in the Göttingen minipig.
Larsen, M O; Rolin, B; Raun, K; Bjerre Knudsen, L; Gotfredsen, C F; Bock, T.
Afiliación
  • Larsen MO; Department of GLP-1 and Obesity Pharmacology, Novo Nordisk A/S, Maaloev, Denmark. mmla@novonordisk.com
Diabetes Obes Metab ; 9 Suppl 2: 170-9, 2007 Nov.
Article en En | MEDLINE | ID: mdl-17919191
Increased knowledge about beta-cell mass and function is important for our understanding of the pathophysiology of type 2 diabetes (T2DM). The relationship between the two is difficult to study in humans, whereas animal models allow studies of consequences of, for example, reduction of beta-cell mass and induction of obesity and procurement of the pancreas for histological examination. An overview of results obtained in the Göttingen minipig in relation to beta-cell function, and mass is provided here. Effects of a primary reduction of beta-cell mass have indicated that not all of the defects of pulsatile insulin secretion in human T2DM can be explained by reduced beta-cell mass. Furthermore, induction of obesity has shown deterioration of beta-cell function and morphological changes in the pancreas. As in humans, obesity leads to an increased beta-cell volume in the minipig, and based on the increased number of islets, neogenesis of islets is an important factor in expansion of beta-cell mass in this species. Measurement of beta-cell function as an estimate of beta-cell mass is, at present, the only method possible in humans, and this approach has been validated using lean and obese minipigs with a range of beta-cell mass. The effects on beta-cell function and mass of obesity of longer duration and/or more pronounced hyperglycaemia remains to be determined, but the models developed so far represent a valuable tool for such investigations.
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Diabetes Mellitus Experimental / Células Secretoras de Insulina / Insulina / Obesidad Tipo de estudio: Prognostic_studies Límite: Animals / Humans Idioma: En Revista: Diabetes Obes Metab Asunto de la revista: ENDOCRINOLOGIA / METABOLISMO Año: 2007 Tipo del documento: Article País de afiliación: Dinamarca Pais de publicación: Reino Unido
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Diabetes Mellitus Experimental / Células Secretoras de Insulina / Insulina / Obesidad Tipo de estudio: Prognostic_studies Límite: Animals / Humans Idioma: En Revista: Diabetes Obes Metab Asunto de la revista: ENDOCRINOLOGIA / METABOLISMO Año: 2007 Tipo del documento: Article País de afiliación: Dinamarca Pais de publicación: Reino Unido