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Chronic myeloproliferative disease induced by site-specific integration of Abelson murine leukemia virus-infected hemopoietic stem cells.
Han, X D; Wong, P M; Chung, S W.
Afiliación
  • Han XD; Morse Institute of Molecular Genetics, Department of Microbiology and Immunology, State University of New York Health Science Center, Brooklyn 11203.
Proc Natl Acad Sci U S A ; 88(22): 10129-33, 1991 Nov 15.
Article en En | MEDLINE | ID: mdl-1682923
We recently showed that hemopoietic stem cells expressing the v-abl oncogene can cause leukemia when injected into lethally irradiated recipient mice. Progenitor cells expressing v-abl did not significantly contribute to disease development, and the leukemia was monoclonal in origin. By serially transplanting v-abl-transduced hemopoietic stem cells into normal, nonirradiated syngeneic recipients, we showed that multiple stem-cell clones do exist in some recipients. These cells fluctuated as normal stem cells do and could home to normal bone marrow. Based on the time course of disease, the recipients developed either an acute or a chronic phase of disorder. All recipients with the acute disease had stem-cell clones with random Abelson murine leukemia virus integration sites. All recipients with the chronic disorder had a specific Abelson murine leukemia virus integration site. We believe this abl-specific integration site, termed ASI, is important in abl-mediated stem-cell leukemogenesis.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Virus de la Leucemia Murina de Abelson / Células Madre Hematopoyéticas / Leucemia Experimental / Trastornos Mieloproliferativos Límite: Animals Idioma: En Revista: Proc Natl Acad Sci U S A Año: 1991 Tipo del documento: Article Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Virus de la Leucemia Murina de Abelson / Células Madre Hematopoyéticas / Leucemia Experimental / Trastornos Mieloproliferativos Límite: Animals Idioma: En Revista: Proc Natl Acad Sci U S A Año: 1991 Tipo del documento: Article Pais de publicación: Estados Unidos