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Identification of proteoglycans as the APRIL-specific binding partners.
Ingold, Karine; Zumsteg, Adrian; Tardivel, Aubry; Huard, Bertrand; Steiner, Quynh-Giao; Cachero, Teresa G; Qiang, Fang; Gorelik, Leonid; Kalled, Susan L; Acha-Orbea, Hans; Rennert, Paul D; Tschopp, Jürg; Schneider, Pascal.
Afiliación
  • Ingold K; Department of Biochemistry, University of Lausanne, CH-1066 Epalinges, Switzerland.
J Exp Med ; 201(9): 1375-83, 2005 May 02.
Article en En | MEDLINE | ID: mdl-15851487
B cell activating factor of the tumor necrosis factor (TNF) family (BAFF) and a proliferation-inducing ligand (APRIL) are closely related ligands within the TNF superfamily that play important roles in B lymphocyte biology. Both ligands share two receptors--transmembrane activator and calcium signal--modulating cyclophilin ligand interactor (TACI) and B cell maturation antigen (BCMA)--that are predominantly expressed on B cells. In addition, BAFF specifically binds BAFF receptor, whereas the nature of a postulated APRIL-specific receptor remains elusive. We show that the TNF homology domain of APRIL binds BCMA and TACI, whereas a basic amino acid sequence (QKQKKQ) close to the NH2 terminus of the mature protein is required for binding to the APRIL-specific "receptor." This interactor was identified as negatively charged sulfated glycosaminoglycan side chains of proteoglycans. Although T cell lines bound little APRIL, the ectopic expression of glycosaminoglycan-rich syndecans or glypicans conferred on these cells a high binding capacity that was completely dependent on APRIL's basic sequence. Moreover, syndecan-1-positive plasma cells and proteoglycan-rich nonhematopoietic cells displayed high specific, heparin-sensitive binding to APRIL. Inhibition of BAFF and APRIL, but not BAFF alone, prevented the survival and/or the migration of newly formed plasma cells to the bone marrow. In addition, costimulation of B cell proliferation by APRIL was only effective upon APRIL oligomerization. Therefore, we propose a model whereby APRIL binding to the extracellular matrix or to proteoglycan-positive cells induces APRIL oligomerization, which is the prerequisite for the triggering of TACI- and/or BCMA-mediated activation, migration, or survival signals.
Asunto(s)

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Proteoglicanos / Proteínas Nucleares / Linfocitos B / Receptores del Factor de Necrosis Tumoral / Proteínas de la Membrana / Modelos Biológicos Tipo de estudio: Diagnostic_studies / Prognostic_studies Límite: Animals / Humans Idioma: En Revista: J Exp Med Año: 2005 Tipo del documento: Article País de afiliación: Suiza Pais de publicación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Proteoglicanos / Proteínas Nucleares / Linfocitos B / Receptores del Factor de Necrosis Tumoral / Proteínas de la Membrana / Modelos Biológicos Tipo de estudio: Diagnostic_studies / Prognostic_studies Límite: Animals / Humans Idioma: En Revista: J Exp Med Año: 2005 Tipo del documento: Article País de afiliación: Suiza Pais de publicación: Estados Unidos