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Genetic screening for a single common LRRK2 mutation in familial Parkinson's disease.
Nichols, William C; Pankratz, Nathan; Hernandez, Dena; Paisán-Ruíz, Coro; Jain, Shushant; Halter, Cheryl A; Michaels, Veronika E; Reed, Terry; Rudolph, Alice; Shults, Clifford W; Singleton, Andrew; Foroud, Tatiana.
Afiliación
  • Nichols WC; Division of Human Genetics, Cincinnati Children's Hospital Medical Center, Cincinnati, OH 45229, USA. Bill.nichols@cchmc.org
Lancet ; 365(9457): 410-2, 2005.
Article en En | MEDLINE | ID: mdl-15680455
Mutations in the leucine-rich repeat kinase 2 (LRRK2) gene cause some forms of autosomal dominant Parkinson's disease. We measured the frequency of a novel mutation (Gly2019 ser) in familial Parkinson's disease by screening genomic DNA of patients and controls. Of 767 affected individuals from 358 multiplex families, 35 (5%) individuals were either heterozygous (34) or homozygous (one) for the mutation, and had typical clinical findings of idiopathic Parkinson's disease. Thus, our results suggest that a single LRRK2 mutation causes Parkinson's disease in 5% of individuals with familial disease. Screening for this mutation should be a component of genetic testing for Parkinson's disease.
Asunto(s)
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Enfermedad de Parkinson / Proteínas Serina-Treonina Quinasas / Mutación Puntual Tipo de estudio: Clinical_trials / Diagnostic_studies / Prognostic_studies / Screening_studies Límite: Adolescent / Adult / Aged / Aged80 / Humans / Male / Middle aged Idioma: En Revista: Lancet Año: 2005 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Enfermedad de Parkinson / Proteínas Serina-Treonina Quinasas / Mutación Puntual Tipo de estudio: Clinical_trials / Diagnostic_studies / Prognostic_studies / Screening_studies Límite: Adolescent / Adult / Aged / Aged80 / Humans / Male / Middle aged Idioma: En Revista: Lancet Año: 2005 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido