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[Effects of 7-oxo-DHEA treatment on the immunoreactivity of BALB/c mice subjected to chronic mild stress].
Liu, Yan-yong; Yang, Nan; Kong, Ling-na; Zuo, Ping-ping.
Afiliación
  • Liu YY; Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences, Peking Union Medical College, Beijing 100005, China.
Yao Xue Xue Bao ; 38(12): 881-4, 2003 Dec.
Article en Zh | MEDLINE | ID: mdl-15040075
AIM: To determine whether 7-oxo-dehydroepiandrosterone (7-oxo-DHEA) can reverse the hypoimmunity in BALB/c mice exposed to chronic mild stress. METHODS: A chronic mild stress animal model was established by subjecting BALB/c mice to a stressful regimen arranged in an unpredicted manner for 4 consecutive weeks. Immunological function alternations under chronic mild stress were assessed by lymphocytes proliferative response to mitogens and NK cell lysis activity test. RESULTS: The studies showed the correlation between the state of depression and abnormalities in the immune response, such as a decrease of T lymphocytes proliferative response to Con A and suppression of cytotoxic of NK cell. Meanwhile, significant decrease of T3 and T4 levels was also observed. When stressed mice were daily given 7-oxo-DHEA 15 mg.kg-1, lymphocyte proliferative response and the NK cell activity were significantly enhanced and the decreased levels of T3 and T4 were restored in the stressed mice. CONCLUSION: 7-oxo-DHEA can improve the depressive symptoms and hypoimmunity of BALB/c mice induced by chronic mild stress as its parent DHEA.
Asunto(s)
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Estrés Fisiológico / Linfocitos T / Deshidroepiandrosterona Límite: Animals Idioma: Zh Revista: Yao Xue Xue Bao Año: 2003 Tipo del documento: Article País de afiliación: China Pais de publicación: China
Buscar en Google
Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Estrés Fisiológico / Linfocitos T / Deshidroepiandrosterona Límite: Animals Idioma: Zh Revista: Yao Xue Xue Bao Año: 2003 Tipo del documento: Article País de afiliación: China Pais de publicación: China