Pathophysiology of tinnitus.

Møller, Aage R

Møller, Aage R.

Afiliación

Møller AR; Callier Center for Communication Disorders, University of Texas at Dallas, School of Human Development, 1966 Inwood Road, Dallas, TX 75235, USA. amoller@utdallas.edu

Otolaryngol Clin North Am ; 36(2): 249-66, v-vi, 2003 Apr.

Article en En | MEDLINE | ID: mdl-12856295

RESUMEN

Tinnitus is not a single entity but a rather diverse group of disorders. Despite symptoms that indicate the ear is the site of the pathology, there is strong evidence that most forms of severe tinnitus are caused by functional changes in the central nervous system. The changes are induced through expression of neural plasticity, some of which may have been caused initially by abnormalities in the ear or the auditory nerve. The involvement of the nonclassical ascending auditory pathway with its subcortical connections to limbic structures (the amygdala) may explain some of the symptoms of some forms of tinnitus including hyperacusis and affective disorders, such as phonophobia and depression, which often accompany severe tinnitus.

Asunto(s)

Acúfeno/fisiopatología; Vías Auditivas/fisiopatología; Sistema Nervioso Central/fisiopatología; Humanos; Plasticidad Neuronal; Acúfeno/etiología

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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Acúfeno Tipo de estudio: Etiology_studies Límite: Humans Idioma: En Revista: Otolaryngol Clin North Am Año: 2003 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos

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