Your browser doesn't support javascript.
loading
The pleckstrin homology domain of phospholipase C-beta2 as an effector site for Rac.
Snyder, Jason T; Singer, Alex U; Wing, Michele R; Harden, T Kendall; Sondek, John.
Afiliación
  • Snyder JT; Department of Pharmacology, The University of North Carolina, Chapel Hill, North Carolina 27599, USA.
J Biol Chem ; 278(23): 21099-104, 2003 Jun 06.
Article en En | MEDLINE | ID: mdl-12657629
Increasing evidence links the activation of Rho family GTPases to the stimulation of lipid hydrolysis catalyzed by phospholipase C (PLC)-beta isozymes. To better define this relationship, members of a library of recombinant Rho GTPases were screened for their capacity to directly engage various purified PLC-beta isozymes. Of the 17 tested members of the Rho family, only the active isoforms of Rac (Rac1, Rac2, and Rac3) both stimulate PLC-beta activity in vivo and bind PLC-beta2 and PLC-beta3, but not PLC-beta1, in vitro. Furthermore, the recognition site for Rac GTPases was localized to the pleckstrin homology (PH) domain of PLC-beta2, and this PH domain is fully sufficient to selectively interact with the active versions of the Rac GTPases, but not with other similar Rho GTPases. Together, these findings present a quantitative evaluation of the direct interactions between Rac GTPases and PLC-beta isozymes and define a novel role for the PH domain of PLC-beta2 as a putative effector site for Rac GTPases.
Asunto(s)
Buscar en Google
Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Fosfolipasas de Tipo C / Fosfoproteínas / Proteínas Sanguíneas / Proteína de Unión al GTP rac1 / Isoenzimas Límite: Animals / Humans Idioma: En Revista: J Biol Chem Año: 2003 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos
Buscar en Google
Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Fosfolipasas de Tipo C / Fosfoproteínas / Proteínas Sanguíneas / Proteína de Unión al GTP rac1 / Isoenzimas Límite: Animals / Humans Idioma: En Revista: J Biol Chem Año: 2003 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos