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HIV-1 Nef associated PAK and PI3-kinases stimulate Akt-independent Bad-phosphorylation to induce anti-apoptotic signals.
Wolf, D; Witte, V; Laffert, B; Blume, K; Stromer, E; Trapp, S; d'Aloja, P; Schürmann, A; Baur, A S.
Afiliación
  • Wolf D; Department of Dermatology Erlangen, University of Erlangen/Nürnberg, Erlangen, Germany.
Nat Med ; 7(11): 1217-24, 2001 Nov.
Article en En | MEDLINE | ID: mdl-11689886
A highly conserved signaling property of Nef proteins encoded by human or simian immunodeficiency virus is the binding and activation of a PAK kinase whose function is unclear. Here we show that Nef-mediated p21-activated kinase (PAK) activation involves phosphatidylinositol 3-kinase, which acts upstream of PAK and is bound and activated by Nef similar to the manner of Polyoma virus middle T antigen. The Nef-associated phosphatidylinositol-3-PAK complex phosphorylated the pro-apoptotic Bad protein without involving the protein kinase B-Akt kinase, which is generally believed to inactivate Bad by serine phosphorylation. Consequently, Nef, but not a Nef mutant incapable of activating PAK, blocked apoptosis in T cells induced by serum starvation or HIV replication. Nef anti-apoptotic effects are likely a crucial mechanism for viral replication in the host and thus in AIDS pathogenesis.
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Proteínas Portadoras / Productos del Gen nef / VIH-1 / Proteínas Serina-Treonina Quinasas / Fosfatidilinositol 3-Quinasas Tipo de estudio: Risk_factors_studies Límite: Animals / Humans Idioma: En Revista: Nat Med Asunto de la revista: BIOLOGIA MOLECULAR / MEDICINA Año: 2001 Tipo del documento: Article País de afiliación: Alemania Pais de publicación: Estados Unidos
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Proteínas Portadoras / Productos del Gen nef / VIH-1 / Proteínas Serina-Treonina Quinasas / Fosfatidilinositol 3-Quinasas Tipo de estudio: Risk_factors_studies Límite: Animals / Humans Idioma: En Revista: Nat Med Asunto de la revista: BIOLOGIA MOLECULAR / MEDICINA Año: 2001 Tipo del documento: Article País de afiliación: Alemania Pais de publicación: Estados Unidos