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Thyroid hormone induces cerebellar astrocytes and C6 glioma cells to secrete mitogenic growth factors.
Trentin, A G; Alvarez-Silva, M; Moura Neto, V.
Afiliación
  • Trentin AG; Departamento de Biologia Celular, Embriologia e Genética, Centro de Ciências Biológicas, Universidade Federal de Santa Catarina, 88040-900 Florianópolis, Santa Catarina, Brazil.
Am J Physiol Endocrinol Metab ; 281(5): E1088-94, 2001 Nov.
Article en En | MEDLINE | ID: mdl-11595667
In this study, the effect of thyroid hormone (triiodothyronine, T(3)) on the secretion of mitogenic growth factors in astrocytes and C6 glioma cells was examined. The proliferating activity of T(3) could be due, at least in part, to the astrocyte secretion of acidic and basic fibroblast growth factor (aFGF and bFGF), tumor necrosis factor-beta, and transforming growth factor-beta. In contrast, the conditioned medium (CM) of T(3)-treated C6 cells was mitogenic to this cell line only after hyaluronidase digestion, suggesting the impairment of growth factor mitogenic activity by hyaluronic acid. Furthermore, the presence of bFGF was significantly greater in the CM of both T(3)-treated astrocytes and T(3)-treated C6 cells than in the corresponding control CM. These data show that T(3) induces cerebellar astrocytes to secrete mitogenic growth factors, predominantly bFGF, that could influence astrocyte and neuronal proliferation via autocrine and paracrine pathways.
Asunto(s)
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Triyodotironina / Cerebelo / Astrocitos / Sustancias de Crecimiento / Glioma Límite: Animals Idioma: En Revista: Am J Physiol Endocrinol Metab Asunto de la revista: ENDOCRINOLOGIA / FISIOLOGIA / METABOLISMO Año: 2001 Tipo del documento: Article País de afiliación: Brasil Pais de publicación: Estados Unidos
Buscar en Google
Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Triyodotironina / Cerebelo / Astrocitos / Sustancias de Crecimiento / Glioma Límite: Animals Idioma: En Revista: Am J Physiol Endocrinol Metab Asunto de la revista: ENDOCRINOLOGIA / FISIOLOGIA / METABOLISMO Año: 2001 Tipo del documento: Article País de afiliación: Brasil Pais de publicación: Estados Unidos