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EGR2 mutation R359W causes a spectrum of Dejerine-Sottas neuropathy.
Boerkoel, C F; Takashima, H; Bacino, C A; Daentl, D; Lupski, J R.
Afiliación
  • Boerkoel CF; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas 77030, USA.
Neurogenetics ; 3(3): 153-7, 2001 Jul.
Article en En | MEDLINE | ID: mdl-11523566
Heterozygous mutations in the early growth response gene 2 (EGR2), which encodes a zinc-finger transcription factor that regulates the late stages of myelination, cause myelinopathies including congenital hypomyelinating neuropathy, Dejerine-Sottas neuropathy (DSN), and Charcot-Marie-Tooth disease type 1. We screened 170 unrelated neuropathy patients without mutations involving the peripheral myelin protein 22 gene (PMP22), the myelin protein zero gene (MPZ), or the gap junction protein beta1 gene (GJB1) and identified two DSN patients with the heterozygous mutation R359W in the alpha-helix domain of the first zinc-finger of EGR2. We now report that this mutation is a recurrent cause of DSN, and that expressivity ranges from that typical for DSN to a more rapidly progressive neuropathy that can cause death by age 6 years. Furthermore, in contrast to patients with typical DSN, patients with the EGR2 R359W mutation have more respiratory compromise and cranial nerve involvement.
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Factores de Transcripción / Neuropatía Hereditaria Motora y Sensorial / Proteínas de Unión al ADN Tipo de estudio: Etiology_studies / Prognostic_studies Límite: Female / Humans / Male Idioma: En Revista: Neurogenetics Asunto de la revista: GENETICA / NEUROLOGIA Año: 2001 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Factores de Transcripción / Neuropatía Hereditaria Motora y Sensorial / Proteínas de Unión al ADN Tipo de estudio: Etiology_studies / Prognostic_studies Límite: Female / Humans / Male Idioma: En Revista: Neurogenetics Asunto de la revista: GENETICA / NEUROLOGIA Año: 2001 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos