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Hypoxic induction of adrenomedullin in cultured human umbilical vein endothelial cells.
Ogita, T; Hashimoto, E; Yamasaki, M; Nakaoka, T; Matsuoka, R; Kira, Y; Fujita, T.
Afiliación
  • Ogita T; Department of Internal Medicine, School of Medicine, University of Tokyo, Japan. ogita-tky@umin.ac.jp
J Hypertens ; 19(3 Pt 2): 603-8, 2001 Mar.
Article en En | MEDLINE | ID: mdl-11327636
OBJECTIVES: The current study evaluated the hypoxic induction of adrenomedullin gene expression and secretion, and its mechanism in cultured human umbilical vein endothelial cells (HUVEC). METHODS: HUVEC were exposed to hypoxia or normoxia as controls for 1 to 24 h. Using Northern blot analysis and a radioimmunoassay, we evaluated adrenomedullin expression in HUVEC. The transcriptional component of adrenomedullin gene regulation was assessed by nuclear run-off experiments, and adrenomedullin mRNA half-life was measured by actinomycin D experiments. RESULTS: We found that hypoxic conditions (1-3% oxygen) significantly increased adrenomedullin mRNA and protein in HUVEC. This increase was inversely proportional to oxygen tension and was reversible upon re-exposure to a 21% oxygen environment Nuclear run-off experiments revealed the enhanced transcriptional rate of adrenomedullin gene. Next, actinomycin D experiments revealed the enhanced adrenomedullin mRNA stability. CONCLUSIONS: These results indicate that hypoxia increases adrenomedullin gene expression and secretion in HUVEC by transcriptional and post-transcriptional mechanisms. Hypoxic induction of adrenomedullin may play a pathophysiological role in the vascular systems.
Asunto(s)
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Péptidos / Venas Umbilicales / Endotelio Vascular / Hipoxia Límite: Humans Idioma: En Revista: J Hypertens Año: 2001 Tipo del documento: Article País de afiliación: Japón Pais de publicación: Países Bajos
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Péptidos / Venas Umbilicales / Endotelio Vascular / Hipoxia Límite: Humans Idioma: En Revista: J Hypertens Año: 2001 Tipo del documento: Article País de afiliación: Japón Pais de publicación: Países Bajos