Your browser doesn't support javascript.
loading
Cytokine responses of oral epithelial cells to Porphyromonas gingivalis infection.
Sandros, J; Karlsson, C; Lappin, D F; Madianos, P N; Kinane, D F; Papapanou, P N.
Afiliación
  • Sandros J; Department of Oral Pathology, Faculty of Odontology, Göteborg University, Sweden. sandros@odontologi.gu.se
J Dent Res ; 79(10): 1808-14, 2000 Oct.
Article en En | MEDLINE | ID: mdl-11077999
Accumulating evidence indicates that epithelia are not merely mechanical barriers but also important elements of the innate immune system. The present study was performed to examine cytokine responses of oral epithelial cells after infection with the periodontal pathogen Porphyromonas gingivalis. The KB-cell line and primary cultures of periodontal pocket epithelium were infected with P. gingivalis for assessment of bacterial invasion by an antibiotic protection assay, and examination of expression of interleukin-1 beta, interleukin-6, interleukin-8, and tumor necrosis factor-alpha by in situ hybridization and immunohistochemistry. We observed that P. gingivalis induces a strong cytokine response, positively correlated with the adhesive/invasive potential of the infecting strain, in both KB cells and primary cultures. These findings indicate that the epithelial cells of the periodontal pocket are an integral part of the immune system, eliciting cytokine responses to a bacterial challenge. In this context, the adhesive/invasive phenotype of P. gingivalis appears to contribute to pathogenicity.
Asunto(s)
Buscar en Google
Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Citocinas / Porphyromonas gingivalis / Células Epiteliales Límite: Humans Idioma: En Revista: J Dent Res Año: 2000 Tipo del documento: Article País de afiliación: Suecia Pais de publicación: Estados Unidos
Buscar en Google
Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Citocinas / Porphyromonas gingivalis / Células Epiteliales Límite: Humans Idioma: En Revista: J Dent Res Año: 2000 Tipo del documento: Article País de afiliación: Suecia Pais de publicación: Estados Unidos