Your browser doesn't support javascript.
loading
Mazindol treatment of negative symptoms.
Carpenter, W T; Breier, A; Buchanan, R W; Kirkpatrick, B; Shepard, P; Weiner, E.
Afiliación
  • Carpenter WT; Maryland Psychiatric Research Center, Department of Psychiatry, University of Maryland, Baltimore, MD 21247, USA.
Neuropsychopharmacology ; 23(4): 365-74, 2000 Oct.
Article en En | MEDLINE | ID: mdl-10989263
Hypodopaminergic and hyponoradrenergic pathophysiology may be a basis for primary and/or secondary negative symptoms in schizophrenia. The hypothesis that enhanced neurotransmission in these systems would be therapeutic for negative symptoms was tested by comparing mazindol and placebo in a double-blind, cross-over design trial. Outcome following mazindol supplementation was comparable to placebo supplementation (F(1,30) = 0.9; p = .57). Results for deficit and non-deficit schizophrenia subjects were similar, and were not affected by whether concurrent the antipsychotic drug treatment was clozapine, fluphenazine, or haloperidol. The efficacy hypothesis was not supported for either primary or secondary negative symptoms.
Asunto(s)
Buscar en Google
Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Escalas de Valoración Psiquiátrica / Trastornos Psicóticos / Esquizofrenia / Inhibidores de Captación de Dopamina / Mazindol Tipo de estudio: Clinical_trials / Diagnostic_studies Límite: Adult / Female / Humans / Male / Middle aged Idioma: En Revista: Neuropsychopharmacology Asunto de la revista: NEUROLOGIA / PSICOFARMACOLOGIA Año: 2000 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido
Buscar en Google
Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Escalas de Valoración Psiquiátrica / Trastornos Psicóticos / Esquizofrenia / Inhibidores de Captación de Dopamina / Mazindol Tipo de estudio: Clinical_trials / Diagnostic_studies Límite: Adult / Female / Humans / Male / Middle aged Idioma: En Revista: Neuropsychopharmacology Asunto de la revista: NEUROLOGIA / PSICOFARMACOLOGIA Año: 2000 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido