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Association and linkage studies of CRH and PENK genes in bipolar disorder: a collaborative IGSLI study.
Am J Med Genet ; 96(2): 178-81, 2000 Apr 03.
Article en En | MEDLINE | ID: mdl-10893493
Corticotropin-releasing hormone (CRH) and proenkephalin (PENK) are hypothalamic peptides involved in the stress response and hypothalamic-pituitary axis regulation. Previous research has implicated these peptides in the pathogenesis of affective disorders. In this study we investigated two polymorphisms located in the genes that code for CRH and PENK by means of association and linkage analyses. A total of 138 bipolar patients and 108 controls were included in the association study. In addition, 24 families were available for linkage analysis, including six families of probands with documented periodic positivity of dexamethasone suppression tests (DST) during remission. We found no association of bipolar disorder with either gene. Similarly, we did not find any evidence of linkage (P = 0.56 for CRH and 0.52 for PENK) in the entire sample or in the subsample of families of DST positive probands. In conclusion, our study does not support the hypothesis that genes coding for CRH or PENK contribute to the genetic susceptibility to bipolar disorder. Am. J. Med. Genet. (Neuropsychiatr. Genet.) 96:178-181, 2000.
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Precursores de Proteínas / Trastorno Bipolar / Hormona Liberadora de Corticotropina / Encefalinas / Ligamiento Genético Tipo de estudio: Clinical_trials / Etiology_studies / Risk_factors_studies Límite: Adult / Female / Humans / Male / Middle aged Idioma: En Revista: Am J Med Genet Año: 2000 Tipo del documento: Article País de afiliación: Canadá Pais de publicación: Estados Unidos
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Precursores de Proteínas / Trastorno Bipolar / Hormona Liberadora de Corticotropina / Encefalinas / Ligamiento Genético Tipo de estudio: Clinical_trials / Etiology_studies / Risk_factors_studies Límite: Adult / Female / Humans / Male / Middle aged Idioma: En Revista: Am J Med Genet Año: 2000 Tipo del documento: Article País de afiliación: Canadá Pais de publicación: Estados Unidos