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Structure-directed discovery of potent non-peptidic inhibitors of human urokinase that access a novel binding subsite.
Nienaber, V L; Davidson, D; Edalji, R; Giranda, V L; Klinghofer, V; Henkin, J; Magdalinos, P; Mantei, R; Merrick, S; Severin, J M; Smith, R A; Stewart, K; Walter, K; Wang, J; Wendt, M; Weitzberg, M; Zhao, X; Rockway, T.
Afiliación
  • Nienaber VL; Department of Structural Biology, Abbott Laboratories, IL 60064-6098, USA. vicki.nienaber@abbott.com.
Structure ; 8(5): 553-63, 2000 May 15.
Article en En | MEDLINE | ID: mdl-10801494
BACKGROUND: Human urokinase-type plasminogen activator has been implicated in the regulation and control of basement membrane and interstitial protein degradation. Because of its role in tissue remodeling, urokinase is a central player in the disease progression of cancer, making it an attractive target for design of an anticancer clinical agent: Few urokinase inhibitors have been described, which suggests that discovery of such a compound is in the early stages. Towards integrating structural data into this process, a new human urokinase crystal form amenable to structure-based drug design has been used to discover potent urokinase inhibitors. RESULTS: On the basis of crystallographic data, 2-naphthamidine was chosen as the lead scaffold for structure-directed optimization. This co-crystal structure shows the compound binding at the primary specificity pocket of the trypsin-like protease and at a novel binding subsite that is accessible from the 8-position of 2-napthamidine. This novel subsite was characterized and used to design two compounds with very different 8-substituents that inhibit urokinase with K(i) values of 30-40 nM. CONCLUSIONS: Utilization of a novel subsite yielded two potent urokinase inhibitors even though this site has not been widely used in inhibitor optimization with other trypsin-like proteases, such as those reported for thrombin or factor Xa. The extensive binding pockets present at the substrate-binding groove of these other proteins are blocked by unique insertion loops in urokinase, thus necessitating the utilization of additional binding subsites. Successful implementation of this strategy and characterization of the novel site provides a significant step towards the discovery of an anticancer clinical agent.
Asunto(s)
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Activador de Plasminógeno de Tipo Uroquinasa / Diseño de Fármacos / Modelos Moleculares / Inhibidores Enzimáticos / Naftalenos Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Revista: Structure Asunto de la revista: BIOLOGIA MOLECULAR / BIOQUIMICA / BIOTECNOLOGIA Año: 2000 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Activador de Plasminógeno de Tipo Uroquinasa / Diseño de Fármacos / Modelos Moleculares / Inhibidores Enzimáticos / Naftalenos Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Revista: Structure Asunto de la revista: BIOLOGIA MOLECULAR / BIOQUIMICA / BIOTECNOLOGIA Año: 2000 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos