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Effects of mitomycin C and carboplatin pretreatment on multidrug resistance-associated P-glycoprotein expression and on subsequent suppression of tumor growth by doxorubicin and paclitaxel in human metastatic breast cancer xenografted nude mice.
Ihnat, M A; Nervi, A M; Anthony, S P; Kaltreider, R C; Warren, A J; Pesce, C A; Davis, S A; Lariviere, J P; Hamilton, J W.
Afiliación
  • Ihnat MA; Department of Pharmacology and Toxicology, Dartmouth Medical School, Hanover, NH 03755-3835, USA.
Oncol Res ; 11(7): 303-10, 1999.
Article en En | MEDLINE | ID: mdl-10757444
Overexpression of P-glycoprotein (Pgp), multidrug resistance-associated protein (MRP), and several other proteins has been associated with development of multidrug resistance by cancer cells, which represents a significant obstacle to successful treatment by chemotherapy. We had previously demonstrated that a single noncytotoxic dose of mitomycin C (MMC), carboplatin, or one of several other DNA cross-linking agents suppressed mRNA expression of the mdr1 gene coding for Pgp, leading to a subsequent suppression of Pgp protein levels and a concomitant decrease in drug efflux. Pretreatment with MMC led to a 5- to 10-fold decrease in the ED50 for cell killing by a subsequent agent such as the Pgp substrate, doxorubicin, but did not affect killing by the non-Pgp substrate, cisplatin. In this study, we report that MMC and carboplatin each significantly suppressed Pgp protein levels in human MDA-MB-435 cells xenografted as solid tumors into the lateral mammary fat pads of female nude mice, with a similar time course as had previously been observed in cell culture. Pretreatment of mice with MMC or carboplatin 48-72 h prior to receiving either doxorubicin or paclitaxel caused a significantly greater reduction in tumor growth rate compared to either agent alone or the combination given simultaneously. These data suggest that a combination chemotherapy regimen consisting of a DNA cross-linking agent given to modulate the MDR phenotype, followed by a second cytotoxic agent, may be an effective treatment for human patients with de novo or late stage acquired multidrug-resistant malignancies.
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neoplasias de la Mama / Protocolos de Quimioterapia Combinada Antineoplásica / Carboplatino / Mitomicina / Miembro 1 de la Subfamilia B de Casetes de Unión a ATP / Resistencia a Múltiples Medicamentos / Proteínas de Neoplasias Tipo de estudio: Risk_factors_studies Límite: Animals / Female / Humans Idioma: En Revista: Oncol Res Asunto de la revista: NEOPLASIAS Año: 1999 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neoplasias de la Mama / Protocolos de Quimioterapia Combinada Antineoplásica / Carboplatino / Mitomicina / Miembro 1 de la Subfamilia B de Casetes de Unión a ATP / Resistencia a Múltiples Medicamentos / Proteínas de Neoplasias Tipo de estudio: Risk_factors_studies Límite: Animals / Female / Humans Idioma: En Revista: Oncol Res Asunto de la revista: NEOPLASIAS Año: 1999 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos