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Role of spleen macrophages in the clearance of scrapie agent early in pathogenesis.
Beringue, V; Demoy, M; Lasmézas, C I; Gouritin, B; Weingarten, C; Deslys, J P; Andreux, J P; Couvreur, P; Dormont, D.
Afiliación
  • Beringue V; CEA, Service de Neurovirologie, DRM/DSV, CRSSA B.P. 6, 92265 Fontenay aux Roses Cedex, France.
J Pathol ; 190(4): 495-502, 2000 Mar.
Article en En | MEDLINE | ID: mdl-10700001
The involvement of spleen macrophages in the early stages of scrapie pathogenesis was studied by applying the 'macrophage-suicide technique' to scrapie-infected mice. This method comprises critically the intravenous administration to mice of dichloromethylene disphosphonate encapsulated into liposomes. Depletion of spleen macrophages before scrapie infection induced an increased amount of scrapie inoculum in the spleen, consequently leading to accelerated scrapie agent replication in the early phase of pathogenesis, as followed by PrPres accumulation, a specific hallmark of scrapie. The same effect was observed when spleen macrophages were depleted just before the beginning of scrapie agent replication. These findings suggest that macrophages may partly control scrapie infection in peripheral tissues by sequestration of the scrapie inoculum and may thus impair early scrapie agent replication in the spleen. In addition to macrophages, most follicular dendritic cells and B lymphocytes, which are thought to support scrapie agent replication, were also transiently depleted by dichloromethylene disphosphonate administration. This suggests that a compensatory mechanism is sufficient to ensure the persistence of infection in these early stages of pathogenesis.
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Scrapie / Bazo / Macrófagos Tipo de estudio: Etiology_studies Límite: Animals Idioma: En Revista: J Pathol Año: 2000 Tipo del documento: Article País de afiliación: Francia Pais de publicación: Reino Unido
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Scrapie / Bazo / Macrófagos Tipo de estudio: Etiology_studies Límite: Animals Idioma: En Revista: J Pathol Año: 2000 Tipo del documento: Article País de afiliación: Francia Pais de publicación: Reino Unido