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Phosphatidylinositol 4-phosphate 5-kinase alpha is a downstream effector of the small G protein ARF6 in membrane ruffle formation.
Honda, A; Nogami, M; Yokozeki, T; Yamazaki, M; Nakamura, H; Watanabe, H; Kawamoto, K; Nakayama, K; Morris, A J; Frohman, M A; Kanaho, Y.
Afiliación
  • Honda A; Department of Biological Information, Tokyo Institute of Technology, Yokohama, Japan.
Cell ; 99(5): 521-32, 1999 Nov 24.
Article en En | MEDLINE | ID: mdl-10589680
Synthesis of phosphatidylinositol 4,5-bisphosphate [PI(4,5)P2], a signaling phospholipid, is primarily carried out by phosphatidylinositol 4-phosphate 5-kinase [PI(4)P5K], which has been reported to be regulated by RhoA and Rac1. Unexpectedly, we find that the GTPgammaS-dependent activator of PI(4)P5Kalpha is the small G protein ADP-ribosylation factor (ARF) and that the activation strictly requires phosphatidic acid, the product of phospholipase D (PLD). In vivo, ARF6, but not ARF1 or ARF5, spatially coincides with PI(4)P5Kalpha. This colocalization occurs in ruffling membranes formed upon AIF4 and EGF stimulation and is blocked by dominant-negative ARF6. PLD2 similarly translocates to the ruffles, as does the PH domain of phospholipase Cdelta1, indicating locally elevated PI(4,5)P2. Thus, PI(4)P5Kalpha is a downstream effector of ARF6 and when ARF6 is activated by agonist stimulation, it triggers recruitment of a diverse but interactive set of signaling molecules into sites of active cytoskeletal and membrane rearrangement.
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Membrana Celular / Fosfotransferasas (Aceptor de Grupo Alcohol) / Fosfatidilinositol 4,5-Difosfato / Factores de Ribosilacion-ADP Límite: Animals Idioma: En Revista: Cell Año: 1999 Tipo del documento: Article País de afiliación: Japón Pais de publicación: Estados Unidos
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Membrana Celular / Fosfotransferasas (Aceptor de Grupo Alcohol) / Fosfatidilinositol 4,5-Difosfato / Factores de Ribosilacion-ADP Límite: Animals Idioma: En Revista: Cell Año: 1999 Tipo del documento: Article País de afiliación: Japón Pais de publicación: Estados Unidos