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Molecular cloning and biological characterization of NK cell activation-inducing ligand, a counterstructure for CD48.
Kubin, M Z; Parshley, D L; Din, W; Waugh, J Y; Davis-Smith, T; Smith, C A; Macduff, B M; Armitage, R J; Chin, W; Cassiano, L; Borges, L; Petersen, M; Trinchieri, G; Goodwin, R G.
Afiliación
  • Kubin MZ; Department of Immunobiology, Immunex Corporation, Seattle, USA. mkubin@immunex.com
Eur J Immunol ; 29(11): 3466-77, 1999 11.
Article en En | MEDLINE | ID: mdl-10556801
Using the monoclonal antibody C1.7, which recognizes a signaling, membrane-bound molecule on human NK and a proportion of CD8(+) T cells, we cloned a novel molecule we refer to as NK cell activation-inducing ligand (NAIL). It is a 365-amino acid protein that belongs to the immunoglobulin-like superfamily with closest homology to murine 2B4, and human CD84 and CD48. Using a soluble NAIL-Fc fusion protein, we determined the counterstructure for NAIL, CD48, which it binds with high affinity. Stimulation of human B cells with recombinant NAIL in the presence of a suboptimal concentration of human CD40 ligand or IL-4 resulted in increased proliferation. Treatment of human dendritic cells with soluble NAIL-leucine zipper protein resulted in an increased release of IL-12 and TNF-alpha. Using recombinant CD48 protein, we demonstrated the ability of this molecule to increase NK cell cytotoxicity and induce IFN-gamma production. We also showed that 2B4 binds to mouse CD48, suggesting that interaction of these receptors may play a similar role in both species. Taken together these results indicate that the NAIL-CD48 interaction may be an important mechanism regulating a variety of immune responses.
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Células Asesinas Naturales / Antígenos CD / Proteínas de la Membrana Límite: Animals / Humans Idioma: En Revista: Eur J Immunol Año: 1999 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Alemania
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Células Asesinas Naturales / Antígenos CD / Proteínas de la Membrana Límite: Animals / Humans Idioma: En Revista: Eur J Immunol Año: 1999 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Alemania