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Detection of collagenase-induced damage of collagen by 9A4, a monoclonal C-terminal neoepitope antibody.
Otterness, I G; Downs, J T; Lane, C; Bliven, M L; Stukenbrok, H; Scampoli, D N; Milici, A J; Mézes, P S.
Afiliación
  • Otterness IG; Department of Cancer, Immunology, and Infectious Diseases, Pfizer Central Research Division, Pfizer Inc, Groton, CT 06340, USA. otteri@pfizer.com
Matrix Biol ; 18(4): 331-41, 1999 Aug.
Article en En | MEDLINE | ID: mdl-10517180
To determine whether the collagen network is compromised by collagenase during acute inflammation, a monoclonal antibody (9A4) was developed with specificity for the C-terminal neoepitope sequence generated by collagenase-cleavage of type II collagen (Gly-Pro-Pro-Gly-Pro-Gln-Gly-COOH). 9A4 was shown to detect the collagen collagenase-cleavage neoepitope with a K = 1.7 x 10(-7) M (type II) and K = 2 x 10(-6) M (type I). It does not recognize uncleaved native or denatured collagen. Articular cartilage from control animals is unstained by 9A4. During acute inflammation elicited in hamsters by intra-articular LPS, positive staining for the 9A4 neoepitope indicated the collagen was damaged. Wheel running exercise was used to apply stress to control cartilage and cartilage from animals with damaged collagen. After 6 months of running, the cartilage from normal animals was unaffected. By contrast, in the group with damaged collagen, the cartilage was fibrillated in all animals and in half of those, the cartilage failed and bony eburnation resulted.
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Colágeno / Colagenasas / Anticuerpos Monoclonales Tipo de estudio: Diagnostic_studies Límite: Animals Idioma: En Revista: Matrix Biol Asunto de la revista: BIOLOGIA MOLECULAR / BIOQUIMICA Año: 1999 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Países Bajos
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Colágeno / Colagenasas / Anticuerpos Monoclonales Tipo de estudio: Diagnostic_studies Límite: Animals Idioma: En Revista: Matrix Biol Asunto de la revista: BIOLOGIA MOLECULAR / BIOQUIMICA Año: 1999 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Países Bajos