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Carbamazepine-induced upregulation of adenosine A1-receptors in astrocyte cultures affects coupling to the phosphoinositol signaling pathway.
Biber, K; Fiebich, B L; Gebicke-Härter, P; van Calker, D.
Afiliación
  • Biber K; Department of Psychiatry, University of Freiburg, Germany.
Neuropsychopharmacology ; 20(3): 271-8, 1999 Mar.
Article en En | MEDLINE | ID: mdl-10063487
The anticonvulsant and antibipolar drug carbamazepine (CBZ) is known to act as a specific antagonist at adenosine A1-receptors. After a 3-week application of CBZ, A1-receptors are upregulated in the rat brain. We have investigated the consequences of this upregulation for the A1-receptor-mediated signal transduction in primary astrocyte cultures from different regions of the rat brain. CBZ treatment for 10 days had no effect on adenosine A1-receptor mRNA expression in cultures with high basal A1-receptor mRNA levels, but increased A1-receptor mRNA in cultures exhibiting low basal A1-receptor mRNA levels. This upregulation of A1-receptor mRNA was accompanied by an upregulation or induction of A1-receptor-mediated potentiation of PLC activity, a property that was not found in these cultures before CBZ treatment. Thus, CBZ treatment for 10 days induces a new quality of adenosine A1-receptor-mediated signal transduction in cells that express low basal A1-receptor numbers.
Asunto(s)
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Fosfatidilinositoles / Carbamazepina / Transducción de Señal / Astrocitos / Receptores Purinérgicos P1 Límite: Animals Idioma: En Revista: Neuropsychopharmacology Asunto de la revista: NEUROLOGIA / PSICOFARMACOLOGIA Año: 1999 Tipo del documento: Article País de afiliación: Alemania Pais de publicación: Reino Unido
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Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Fosfatidilinositoles / Carbamazepina / Transducción de Señal / Astrocitos / Receptores Purinérgicos P1 Límite: Animals Idioma: En Revista: Neuropsychopharmacology Asunto de la revista: NEUROLOGIA / PSICOFARMACOLOGIA Año: 1999 Tipo del documento: Article País de afiliación: Alemania Pais de publicación: Reino Unido