RESUMO

BACKGROUND: Subtalar hyperpronation and ankle dorsiflexion restriction have been theoretically associated with Achilles tendinopathy (AT). However, evidence to support these associations is lacking. OBJECTIVES: To compare foot alignment and ankle dorsiflexion range of motion (ROM) between the symptomatic and non-symptomatic limbs of individuals with unilateral AT. And to verify whether differences exist between individuals with symptomatic pronated feet and individuals with symptomatic neutral/supinated feet in terms of tendon pain, structure, and symptom severity. METHODS: Sixty-three participants with unilateral AT underwent a bilateral evaluation of pain during tendon palpation, symptom severity, tendon thickening, tendon neovascularization, ankle dorsiflexion ROM, and foot posture alignment [foot posture index (FPI), navicular drop, navicular drift, and longitudinal arch angle (LAA)]. Side and group comparisons were made using t-tests and correlations were evaluated using the Pearson test. RESULTS: There were no differences between the symptomatic and non-symptomatic limbs regarding foot posture alignment. Specifically, non-significant negligible differences were observed between limbs regarding FPI [mean difference (MD)=-0.23; 95% confidence interval (CI)=-0.70, 0.25), navicular drop (MD=0.58 mm; 95%CI=-0.25, 1.43), navicular drift (MD=0.16 mm; 95%CI=-0.77, 1.09), and LAA (MD=0.30º; 95%CI=-1.74, 2.34). There was no difference between limbs regarding ankle dorsiflexion ROM. However, lower ankle dorsiflexion was associated with worse symptom severity (r = 0.223). Finally, no difference was observed between individuals with symptomatic pronated feet and individuals with symptomatic neutral/supinated feet in terms of tendon pain or structure. CONCLUSIONS: Static foot alignment measures do not seem to be clinically relevant in patients with AT. Smaller ankle dorsiflexion ROM, however, was associated with greater symptom severity in this population.

Assuntos

Tendão do Calcâneo , Tendinopatia , Humanos , Tornozelo , Estudos Transversais , Postura , Articulação do Tornozelo , Amplitude de Movimento Articular

RESUMO

Calcific insertional Achilles tendinopathy (CIAT) is a relatively common musculoskeletal entity that results in significant pain and disability, as well as posterior muscle group weakness. There is a lack of evidence criteria to support the timing of operative intervention, choice of procedures, or whether equinus requires treatment. The purpose of this study was to retrospectively review 45 patients (48 feet) who have undergone surgical management of CIAT with concomitant posterior muscle group weakness with the single heel rise testing. All patients underwent debridement and repair of the Achilles tendon with reattachment of the Achilles tendon to the calcaneus, ostectomy of the calcaneus, and flexor hallucis longus tendon transfer. Those patients with equinus also underwent gastrocnemius recession. The focus includes patient-reported satisfaction, time to return to normal shoe gear, and the incidence of revision surgery. The overall average of time to weightbearing was 4.3 weeks. After surgery, 73.3% (n = 33) of the 45 patients responded to the following question: "Would you have this surgery done again?" Of these patients, 93.9% (n = 31) responded "Yes" and 6.1% (n = 2) responded "Unsure." Of the same 33 patients, 84.8% (n = 28) responded that they were "Very Satisfied" with the procedure and 15.2% (n = 5) responded that they were "Satisfied." Twelve patients (26.7%) did not respond to either question. One of the 12 patients (8.3%) who did not respond had bilateral procedures. None of the patients experienced tendon rupture, deep vein thrombosis, or the need for revision surgery. Four patients (8%) experienced a superficial infection, whereas 1 patient (2%) had development of a deep infection. No correlations were found when looking at the relationship between body mass index and return to weightbearing/normal shoe gear with Spearman analysis.

Assuntos

Tendão do Calcâneo/cirurgia , Calcinose/cirurgia , Imageamento por Ressonância Magnética/métodos , Satisfação do Paciente , Tendinopatia/cirurgia , Transferência Tendinosa/métodos , Tendão do Calcâneo/diagnóstico por imagem , Tendão do Calcâneo/fisiopatologia , Adulto , Idoso , Calcâneo/cirurgia , Calcinose/diagnóstico por imagem , Calcinose/fisiopatologia , Estudos de Coortes , Terapia Combinada/métodos , Feminino , Humanos , Masculino , Pessoa de Meia-Idade , Duração da Cirurgia , Osteotomia/métodos , Cuidados Pós-Operatórios/métodos , Cuidados Pré-Operatórios/métodos , Estudos Retrospectivos , Fatores de Risco , Tendinopatia/diagnóstico por imagem , Tendinopatia/fisiopatologia , Resistência à Tração , Resultado do Tratamento

RESUMO

O tendão é formado por um tecido conjuntivo fibroso denso e regular, responsável pela transmissão de forças e pela união do tecido muscular esquelético aos ossos, tanto em sua origem quanto inserção, permitindo, assim, o movimento das articulações e a manutenção da postura corporal. Frequentemente, os tendões são alvos de lesões por diversas causas, sendo elas, degenerativas ou inflamatórias, com diferentes graus de intensidade. As tendinopatias são caracterizadas por uma degeneração das fibras de colágeno, aumento do número de fibroblastos, formação de tecido de granulação e ausência de células inflamatórias. A cicatrização da lesão envolve componentes extrínsecos e intrínsecos, no qual o processo de remodelamento da área lesionada é lento até completa regeneração da estrutura tendínea. Neste contexto, a reabilitação fornece diversos métodos para o auxílio do processo cicatricial tendíneo, através da laserterapia e ultrassom terapêutico, além de atuar no retorno funcional precoce por meio de exercícios terapêuticos e na prevenção de novas lesões. Neste artigo, revisa-se desde a fisiopatologia das lesões tendíneas em pequenos animais, até as principais opções fisiátricas para seus tratamentos e prevenções.

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The tendon is formed from a dense and regular fibrous connective tissue responsible for transmitting forces and by union of skeletal muscle tissue to bones in both its origin and insertion, thus allowing the movement of the joints and the main tenance of body posture. Often, tendon injuries are targets for various reasons, they are, degenerative or inflammatory conditions with different degrees of intensity. Tendinopathies are characterized by a degeneration of collagen fibers, increasing the number of fibroblasts, granulation tissue formation and absence of inflammatory cells. The tendon healing involves extrinsic and intrinsic components. During weeks, the injured area is subjected to a remodeling process, leading to regeneration of tendon structure. In this context, rehabilitation provides several methods for assistance from tendon healing process, such as laser therapy, therapeutic ultrasound, in addition to performing the functional return through exercise therapy and prevention of new lesions. In this article, we revise up from the pathophysiology of tendon lesions in small animals, to the main physical therapy options for their treatments and preventions.

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EI tendón es fqrmado a partir de un tejido conectivo fibroso denso y regular, responsable de las fuerzas de transmisión y poria unión del tejido muscular a los huesos, en su origen y la inserción, loque permite el movimiento de la articulación y el mantenimiento de la postura del cuerpo. A menudo, los tendones son lesiones diana, por diversas razones, que son, condiciones degenerativas o inflamatorias con diferentes grados de intensidad. Tendinopatías se caracterizar, por la degeneración de las fibras de colágeno, aumento en el número de fibroblastos, la formación de tejido de granulación y la ausencia de células inflamatorias. En la citratrización hay componentes intrínsecos y extrínseca, y el proceso de remodelación de la zona lesionada es lento para completar la regeneración de la estructura de tendón. En este contexto, la rehabilitación tiene diversos meios para ayudar al proceso de cicatrización del tendón por la terapia con láser y ultrasonido terapéutico, además de actuar en el retorno funcional temprana por los ejercicios terapéuticos y prevenir una lesión mayor. En este artículo, se revisa a partir de la fisiopatología de las lesiones del tendón en pequenos animales, a las opciones principales fisiátricas para sus tratamientos y prevenciones.

Assuntos

Animais , Manipulações Musculoesqueléticas/veterinária , Modalidades de Fisioterapia/veterinária , Tendinopatia/terapia , Tendinopatia/veterinária , Cicatrização , Colágenos Fibrilares , Fibroblastos/patologia , Terapêutica/veterinária

RESUMO

O tendão é formado por um tecido conjuntivo fibroso denso e regular, responsável pela transmissão de forças e pela união do tecido muscular esquelético aos ossos, tanto em sua origem quanto inserção, permitindo, assim, o movimento das articulações e a manutenção da postura corporal. Frequentemente, os tendões são alvos de lesões por diversas causas, sendo elas, degenerativas ou inflamatórias, com diferentes graus de intensidade. As tendinopatias são caracterizadas por uma degeneração das fibras de colágeno, aumento do número de fibroblastos, formação de tecido de granulação e ausência de células inflamatórias. A cicatrização da lesão envolve componentes extrínsecos e intrínsecos, no qual o processo de remodelamento da área lesionada é lento até completa regeneração da estrutura tendínea. Neste contexto, a reabilitação fornece diversos métodos para o auxílio do processo cicatricial tendíneo, através da laserterapia e ultrassom terapêutico, além de atuar no retorno funcional precoce por meio de exercícios terapêuticos e na prevenção de novas lesões. Neste artigo, revisa-se desde a fisiopatologia das lesões tendíneas em pequenos animais, até as principais opções fisiátricas para seus tratamentos e prevenções.(AU)

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The tendon is formed from a dense and regular fibrous connective tissue responsible for transmitting forces and by union of skeletal muscle tissue to bones in both its origin and insertion, thus allowing the movement of the joints and the main tenance of body posture. Often, tendon injuries are targets for various reasons, they are, degenerative or inflammatory conditions with different degrees of intensity. Tendinopathies are characterized by a degeneration of collagen fibers, increasing the number of fibroblasts, granulation tissue formation and absence of inflammatory cells. The tendon healing involves extrinsic and intrinsic components. During weeks, the injured area is subjected to a remodeling process, leading to regeneration of tendon structure. In this context, rehabilitation provides several methods for assistance from tendon healing process, such as laser therapy, therapeutic ultrasound, in addition to performing the functional return through exercise therapy and prevention of new lesions. In this article, we revise up from the pathophysiology of tendon lesions in small animals, to the main physical therapy options for their treatments and preventions.(AU)

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EI tendón es fqrmado a partir de un tejido conectivo fibroso denso y regular, responsable de las fuerzas de transmisión y poria unión del tejido muscular a los huesos, en su origen y la inserción, loque permite el movimiento de la articulación y el mantenimiento de la postura del cuerpo. A menudo, los tendones son lesiones diana, por diversas razones, que son, condiciones degenerativas o inflamatorias con diferentes grados de intensidad. Tendinopatías se caracterizar, por la degeneración de las fibras de colágeno, aumento en el número de fibroblastos, la formación de tejido de granulación y la ausencia de células inflamatorias. En la citratrización hay componentes intrínsecos y extrínseca, y el proceso de remodelación de la zona lesionada es lento para completar la regeneración de la estructura de tendón. En este contexto, la rehabilitación tiene diversos meios para ayudar al proceso de cicatrización del tendón por la terapia con láser y ultrasonido terapéutico, además de actuar en el retorno funcional temprana por los ejercicios terapéuticos y prevenir una lesión mayor. En este artículo, se revisa a partir de la fisiopatología de las lesiones del tendón en pequenos animales, a las opciones principales fisiátricas para sus tratamientos y prevenciones.(AU)

Assuntos

Animais , Tendinopatia/terapia , Tendinopatia/veterinária , Modalidades de Fisioterapia/veterinária , Manipulações Musculoesqueléticas/veterinária , Terapêutica/veterinária , Colágenos Fibrilares , Cicatrização , Fibroblastos/patologia

RESUMO

Abstract: introduction: The temporalis muscle plays an essential role in mastication and is actively involved in the mandibular closing movement. It is covered by a fibroelastic fascia that forms its tendon. Tendinitis is a degenerative and inflammatory process, which originates in the tendon-bone junction. Signs and symptoms such as swelling, pain, tenderness on palpation, limitation of movement and mouth opening are frequently associated with other temporomandibular disorders and not with tendinitis as a causal factor. Objective: To describe a clinical case identifying the diagnostic process and management of tendinitis of the temporalis muscle. Case report: A 30-year old male patient who sought treatment after continuous squeezing pain in the zygomatic and bilateral temporal regions with increased pain during mouth opening and mandibular function. The patient referred pain in the insertion region of the tendon of the temporalis muscle. Pain was removed after using anesthesia, consequently confirming the diagnosis of tendinitis of the temporalis muscle. Primary management measures were performed and then peritendinous corticosteroids were administered. The patient did not refer spontaneous or functional pain during check-up. Conclusion: Tendinitis of the temporalis muscle is a common condition, although frequently underdiagnosed. A good differential diagnosis must be performed to avoid confusion with other common conditions such as odontogenic pain, sinusitis, arthralgia, myofascial pain and migraine. Management depends on the type of tendinitis. It usually occurs in conjunction with other types of TMD or facial pain, so it is important to know the different clinical characteristics of pathologies with similar manifestations.

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Resumen: introducción: El músculo temporal es fundamental en la masticación, participa activamente en los movimientos de cierre mandibular. Está recubierto por una fascia fibroelástica que conforma su tendón. La tendinitis es un proceso degenerativo e inflamatorio, que se origina en la unión tendón-hueso. Con frecuencia, signos y síntomas como inflamación, dolor a la palpación, limitación del movimiento y apertura, son asociados a otros trastornos temporomandibulares no considerando la tendinitis como un factor causal. Objetivo: Describir un caso clínico identificando el proceso diagnóstico y el manejo de una tendinitis del músculo temporal. Descripción del caso: Paciente sexo masculino de 30 años, acude por dolor opresivo continuo en la región cigomática y temporal bilateral con aumento del dolor en apertura y función mandibular. A la palpación presenta dolor en la zona de inserción del tendón del músculo temporal, el cual se elimina al anestesiar confirmando el diagnóstico de tendinitis del músculo temporal. Se realizan medidas primarias de manejo y luego se infiltra corticoides peritendineos. Al control el paciente no presenta dolor espontáneo ni en función. Conclusión: La tendinitis del músculo temporal es una patología frecuente, aunque subdiagnosticada. Debe realizarse el correcto diagnóstico diferencial con patologías frecuentes como dolor odontogénico, sinusitis, artralgia, dolor miofascial y migraña. El manejo dependerá del tipo de tendinitis. Habitualmente se presenta en conjunto con otros o dolores faciales, por lo que es importante conocer las distintas características clínicas de patologías con presentaciones similares.

Assuntos

Humanos , Masculino , Adulto , Músculo Temporal/patologia , Transtornos da Articulação Temporomandibular/diagnóstico , Transtornos da Articulação Temporomandibular/terapia , Diagnóstico Diferencial

Assuntos

Humanos , Masculino , Feminino , Recém-Nascido , Lactente , Pré-Escolar , Criança , Pediatria , Quinolonas , Fluoroquinolonas/intoxicação , Artropatias , Antibacterianos , Sistema Musculoesquelético

RESUMO

Las lesiones del tendón, denominadas “Tendinosis” son muy frecuentes en la práctica de deportistas de alto rendimiento y recreacionales. Están dentro de las patologías de sobreuso que representan cerca del 60 por ciento de las lesiones deportivas y se relacionan con una sobrecarga cíclica repetidas. Los tendones más afectados del organismo son el rotuliano, aquiliano y manguito rotador. Estudios demuestran que la condición de tendinosis aumenta significativamente su incidencia en relación a la edad; sexo masculino y obesidad. El incremento de la vascularización y terminaciones nerviosas, asociado a un aumento en la producción local de neurotransmisores explicaría la presencia de dolor crónico en las tendinosis. Tendinosis implica una pérdida progresiva de la capacidad de respuesta del tendón para una adecuada regeneración o cicatrización cuando es sometido a una carga cíclica de gran volumen en forma reiterada. La carga cíclica alta repetida induce 1- Estrés oxidativo y apoptosis. 2- Genes cartílago sustituto. Histológicamente esta falla en la respuesta reparativa se refleja en descontrolada proliferación de Tenocitos, disrupción de fibras colágenas, alteraciones en la matriz extracelular, incremento en el fenómeno de apoptosis y cambios en la homeostasis de las Metalo-proteinasas. Se inicia la destrucción de la matriz, aumenta la vascularización y terminaciones nerviosas y el tendón presenta dolor y mecánicamente respuestas a la carga inferiores a un tendón sano. Existe una gran cantidad de opciones de tratamientos pero el objetivo de este artículo es presentar aquellas opciones que nuestro equipo de trabajo aplica, basada en las ciencias básicas y últimos hallazgos con evidencia científica 1- Ejercicios excéntricos asociados a aplicación de ondas de choque radiales han demostrado alto grado de éxito 2- Uso de sustancias esclerosante (polidocanol) y concentrado...

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Tendinopathies account for a substantial proportion of overuse injuries associated with sports1 and are a common cause of disability. Most major tendons, such as the Achilles, patellar, rotator cuff, and forearm extensor tendons (among others), are vulnerable to overuse, which induces pathological changes in the tendon. Tendinopathy has an increased incidence with age and the male gender and with obesity. Excessive long-distance running, intensity, and hill work are risk factors for acute. Tendinopathy is a failed healing response of the tendon. The aim of this review is to identify recent advances in the understanding of tendinopathy, particularly from a cell and molecular biology perspective. There has been much new information and there are many gaps in our understanding of the pathogenesis of tendinopathy. The current hypothesisis that tendinopathy is induced when tendon cells experience a large volume of repetitive load. High doses of cyclical strain induce genes for two major pathways ) oxidative stress- apoptosis; and cartilage like genes. The tendon cells become rounded and apoptotic and produce a matrix that contains less Type I collagen and is more cartilaginous and ‘immature’’ in nature. The therapeutic options with clinical evidence are 1- Combining eccentric training and shock wave therapy produces higher success rates compared with eccentric loading alone or shock wave therapy alone. 2- The use of injectable substances such as platelet-richplasma, autologuos blood, polidocanol, corticosteroids, and aprotinin in and around tendons is popular, but there isminimal clinical evidence to support their use: 3- The aim of operative treatment is to excise fibrotic adhesions, remove areas of failed healing, and make multiple longitudinal incisions in the tendon to detect intratendinous lesions and to restore vascularity and possibly stimulate the remaining viable cells to initiate cell matrix response...

Assuntos

Apoptose , Traumatismos em Atletas , Medicina Esportiva , Tendinopatia/terapia , Transtornos Traumáticos Cumulativos , Ultrassonografia

RESUMO

Introducción: el término tendinitis ha sido usado con frecuencia para referirse a la enfermedad alrededor del epicóndilo, sin embargo, tendinosis sería más apropiado porque el proceso patológico es degenerativo con presencia de fibroblastos e hiperplasia vascular. El tratamiento en general es conservador, menos de 10 por ciento requiere cirugía. Objetivo: describir la anatomía de los músculos en el epicóndilo lateral con el enfoque sobre el extensor carpi radialis brevis (ECRB) y el ligamento colateral lateral. Métodos: se disecaron 20 codos de cadáveres frescos. Se identificó el origen del ECRB y del ligamento colateral lateral; se hicieron mediciones entre ellos y respecto al epicóndilo. Resultados: el origen tendinoso del ECRB se encontró en todos los casos profundo al extensor digitorum comunis. En 19 codos se identificó el tendón del ECRB como una estructura separada, que se originaba anterior al epicóndilo en promedio de 6 mm y con un ancho de 7,93 mm. En todos los codos se observó que el origen del ligamento colateral lateral era distal al origen del ECRB. Conclusión: se observaron dos patrones de distribución proximal del ECRB. En 60 por ciento discurría profundo al extensor digitorum comunis y en 40 por ciento superficial, en el mismo plano que el extensor carpi radialis longus y el extensor digitorum comunis. El ligamento colateral lateral se encontraba en la parte media del cóndilo en 40 por ciento de su diámetro, dato útil en la técnica artroscópica, porque al realizar la fasciotomía profunda del extensor carpi radialis brevis, existe riesgo de seccionar el ligamento debido a la inmediata vecindad entre este y el ligamento(AU)

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Introduction: the term tendinitis has been used frequently for the disease around the epicondyle; however, tendinosis would be more appropriate because of the pathologic process is degenerative with presence of fibroblasts and vascular hyperplasia. Treatment in general is conservative, less of 10 percent require surgery. Objective: to describe the anatomy of muscles in the lateral epicondyle with the focus on the extensor carpi radialis brevis (ECRB) and the lateral collateral ligament. Methods: twenty elbows were dissected from fresh cadavers. The origin of ECRB and of the lateral collateral ligament was identified; measurements between them werw made and regarding the epicondyle. Results: the tendinous origin of ECRB was present in all the cases located deep into the extensor digitorum comunis. In 19 elbows the tendon of the ECRB was identified as a separate structure, originated anterior to epicondyle in 6 mm as average and 7.93 mm in width. In all elbows it was noted that the origin of the lateral collateral ligament was distal to origin of ECRB. Conclusions: there were two patterns of proximal distribution of ECRB. In the 60 percent passed deep to extensor digitorum comunis and in the 40 percent it was superficial in the same plane that the extensor carpi radialis longus and the extensor digitorum comunis. The lateral collateral ligament was located in the middle area of condyle in the 40 percent of its diameter, it is a useful datum for arthroscopy technique because of performing the deep fasciotomy of extensor carpi radialis brevis, there is the risk of to section the ligament due to the immediate proximity between it and the ligament(AU)

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Introduction: le terme tendinite a été fréquemment utilisé pour définir la maladie affectant la région épicondylienne. Cependant, le terme tendinopathie serait le plus approprié, parce que le processus pathologique est dégénératif avec la présence de fibroblastes et hyperplasie vasculaire. Le traitement est généralement conservateur, moins de 10 percent des patients ont besoin d'une chirurgie. Objectifs: décrire l'anatomie des muscles de l'épicondyle latéral, en mettant l'accent sur l'extensor carpi radialis brevis (ECRB) et le ligament collatéral latéral. Méthodes: vingt coudes de cadavres frais ont été disséqués; l'origine de l'ECRB et celle du ligament collatéral ont été identifiées. Résultats: dans tous les cas, on a trouvé que l'origine tendineuse de l'ECRB était profonde par rapport à l'extensor digitorum comunis. Le tendon de l'ECRB a été identifié dans 19 cas, avec une structure séparée, d'origine antérieure par rapport à l'épicondyle de 6 mm, et d'un épaisseur de 7.93 mm. Dans tous les cas, on a également trouvé que l'origine du ligament collatéral était distale par rapport à l'origine de l'ECRB. Conclusions: on a remarqué qu'il y avait deux schémas de distribution proximale de l'ECRB. Soixante pour cent étaient profonds par rapport à l'extensor digitorum comunis, et quarante pour cent étaient superficiels au même plan que l'extensor carpi radialis longus et l'extensor digitorum comunis. Le ligament collatéral latéral se trouvait au milieu du condyle dans 40 percent de son diamètre, donnée utile pour la technique arthroscopique, parce que quand une fasciotomie profonde de l'extensor carpi radialis brevis est réalisée, il y a risque de sectionner le ligament dû à leur proximité(AU)

Assuntos

Humanos , Adulto , Pessoa de Meia-Idade , Cotovelo de Tenista , Cotovelo/anatomia & histologia , Tendinopatia , Cadáver

RESUMO

This study addresses the morphopathogenesis of Achilles tendinosis, using a rat model and presenting quantitative analysis of time-dependent histological changes. Thirty Wistar rats were used, randomly split in experimental and control groups. Animals of the experimental group were submitted to a treadmill running scheme. Five animals of each group were euthanized at four, eight and sixteen weeks. Achilles tendons were collected and processed routinely for histopath sections. Slides were stained by Hematoxylin-Eosin, Picrosirius Red, Alcian Blue, AgNOR, TUNEL and evaluated morphometrically. Cellular density decreased slightly along the time and was higher in the experimental group than in controls at fourth, eighth and sixteenth weeks. Fiber microtearing, percentual of reticular fibers and glycosaminoglycans content increased along the time and were higher in experimental group than in controls at all-time intervals. AgNOR labeling here interpreted as a marker of transcription activity was higher in the experimental groups than in controls at all-time intervals. Apoptotic cells were more frequent and diffusely distributed in tendinosis samples than in control groups. These results suggest that as mechanical overload is becoming chronic, cellular turnover and matrix deposition increases leading to tendinosis. The combination of staining techniques and morphometry used here to describe the evolution of lesions occurring in a rat model system has proved to be suited for the study of induced Achilles tendinosis.

Assuntos

Tendão do Calcâneo/patologia , Transtornos Traumáticos Cumulativos/patologia , Tendinopatia/patologia , Traumatismos dos Tendões/patologia , Tendão do Calcâneo/lesões , Tendão do Calcâneo/metabolismo , Animais , Apoptose , Colágeno/metabolismo , Transtornos Traumáticos Cumulativos/metabolismo , Modelos Animais de Doenças , Glicosaminoglicanos/metabolismo , Marcação In Situ das Extremidades Cortadas , Masculino , Ratos , Ratos Wistar , Corrida , Coloração e Rotulagem , Tendinopatia/metabolismo , Traumatismos dos Tendões/metabolismo , Fatores de Tempo

RESUMO

Supraspinatus tendinosis was recently reported in dogs but it is a well-documented disorder in human beings, specie in which is considered the cause of pain and dysfunction in 51% of cases of shoulder problems. It can cause pain and lameness or be asymptomatic in dogs. The clinical relevance in canine specie is unclear and seems to be underestimated. The aim of this paper is to review the possible etiologies involved in this tendinopathy, the main diagnostic methods applied and actual options of therapies in veterinary medicine. Simultaneously, call the clinicians attention to this disorder as differential diagnosis for forelimb pain and lameness diseases.  

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A tendinose do supra-espinhoso é uma desordem recentemente relatada em cães e bem documentada em seres humanos, espécie na qual representa 51% das causas de alteração na articulação do ombro. Em cães, a afecção pode levar à dor na articulação acometida e claudicação ou permanecer assintomática. O significado clínico do problema ainda não foi definido na espécie canina e parece ser subestimado. O objetivo deste trabalho é discorrer sobre as possíveis etiologias desta tendinopatia e principais métodos diagnósticos, bem como abordar as terapias mais aplicadas atualmente em medicina veterinária, atentando ao clínico sobre as características da doença e a importância de incluí-la no diagnóstico diferencial de causas de dor e claudicação do membro anterior em cães.    

RESUMO

Supraspinatus tendinosis was recently reported in dogs but it is a well-documented disorder in human beings, specie in which is considered the cause of pain and dysfunction in 51% of cases of shoulder problems. It can cause pain and lameness or be asymptomatic in dogs. The clinical relevance in canine specie is unclear and seems to be underestimated. The aim of this paper is to review the possible etiologies involved in this tendinopathy, the main diagnostic methods applied and actual options of therapies in veterinary medicine. Simultaneously, call the clinicians attention to this disorder as differential diagnosis for forelimb pain and lameness diseases.  

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A tendinose do supra-espinhoso é uma desordem recentemente relatada em cães e bem documentada em seres humanos, espécie na qual representa 51% das causas de alteração na articulação do ombro. Em cães, a afecção pode levar à dor na articulação acometida e claudicação ou permanecer assintomática. O significado clínico do problema ainda não foi definido na espécie canina e parece ser subestimado. O objetivo deste trabalho é discorrer sobre as possíveis etiologias desta tendinopatia e principais métodos diagnósticos, bem como abordar as terapias mais aplicadas atualmente em medicina veterinária, atentando ao clínico sobre as características da doença e a importância de incluí-la no diagnóstico diferencial de causas de dor e claudicação do membro anterior em cães.