RESUMO
Field collection of oocytes in mares using transvaginal follicular aspiration (TVA) for embryo production has the potential to revolutionate the equine industry. Protocols for TVA in specialized laboratory settings have been described in the scientific literature since the early 1980s. The objective of this study was to determine the success rate of TVA oocytes recovery under ambulatory conditions. A secondary goal of this study was to determine if TVA is associated with any health complications when performed by recently trained practitioners in the field. Follicles (n = 296) from 66 adult clinically healthy mares were aspirated over a period of 6 days. TVAs were performed by 22 veterinarians with 5-20 years of experience in equine and bovine reproductive medicine, but no previous experience in TVA. Oocytes (n = 145) were recovered. No short- or long-term systemic or local complications were observed following TVA in any of the mares used in this study. Fifty-six out of 66 mares became pregnant within 3 months following TVA. This study shows that with proper training, TVA can be successfully used to obtain equine oocytes with no health complications under field conditions in nonspecialized laboratory settings.
Assuntos
Recuperação de Oócitos , Oócitos , Animais , Bovinos , Embrião de Mamíferos , Feminino , Cavalos , Recuperação de Oócitos/veterinária , GravidezAssuntos
Hemorragia Gastrointestinal/etiologia , Gastroscopia , Gastropatias/etiologia , Estômago/irrigação sanguínea , Malformações Vasculares/diagnóstico por imagem , Adulto , Hemorragia Gastrointestinal/diagnóstico por imagem , Humanos , Masculino , Estômago/diagnóstico por imagem , Gastropatias/diagnóstico por imagem , Malformações Vasculares/complicaçõesRESUMO
El clima social ha sido objeto de estudio en distintos escenarios,como la escuela, la familia, los centros de trabajo olos establecimientos penitenciarios. Este artículo presentalos resultados de la aplicación de la Escala de Clima Socialpara Instituciones Correccionales (CIES) (Moos, Moos &Trickett, 1995) en un establecimiento penitenciario femenilde Aguascalientes, México. El estudio fue psicométrico noexperimental, con cohorte transversal, y en él participaron72 internas, que eran la población total del establecimiento.Con los instrumentos contestados se corrieron estudiosde correlación bivariada, cuyos resultados permitieron identificarque variables como el tipo de delito, el nivel de riesgo,la presencia de algunos diagnósticos psicopatológicos y laescolaridad correlacionan positivamente con la percepciónelevada de algunas subescalas del instrumento usado paraanalizar el clima social. Asimismo, se obtuvo un perfil globalde percepción de este clima, que mostró que, en general,este es percibido como favorable.
The social climate has been subject to scrutiny in differentenvironments such as schools, family groups, work centers,and prisons. This article shows the results of the applicationof the Social Climate Scale for Correctional Institutions (CIES)(Moos, Moos & Trickett, 1995) at a womens correctional centerin Aguascalientes, Mexico. This was a non-experimentalcohort, cross sectional study of psychometrical nature, withthe participation of 72 inmates composing the total femalepopulation of this establishment. With the instruments providedby the answers it was possible to run bivariate correlationstudies, the results of which helped realize that variablessuch as the type of crime, the risk level, the presence ofsome psychopathological diagnosis and the schooling levelare positively correlated with the elevated perception ofsome subscales of the instrument used to analyze the socialclimate. Likewise, with respect to this climate, a global perceptionprofile was obtained, which showed that it is perceivedas favorable in general terms.
O clima social foi objeto do estudo em cenários diferentes,como a escola, a família, os centros de trabalho ou os estabelecimentospenitenciário. Este artigo apresenta os resultadosda aplicação da Escala do Clima Social ara instituiçõesCorrecionales (CIES) (Moos, Moos & Trickett, 1995) em umestabelecimento penitenciário de mulheres de Aguascalientes,México. O estudo foi psicométrico não experimental,com coorte transversal, e nele participaram 72 internas, queera a população total do estabelecimento. Com os instrumentosrespondidos, os estudos de correlação bivariada foramrealizados. Os resultados permitiram identificar quaisvariáveis, como o tipo de crime, o nível do risco, a presençade alguns diagnósticos e a escolaridade correlacionam positivamentecom a percepção elevada de algumas subescalasdo instrumento usado para analisar o clima social. Também,um perfil global da percepção deste clima foi obtido; issomostrou que, me geral, é percebido como favorável.
Assuntos
Humanos , Criminologia , SociologiaRESUMO
La deficiencia de biotinidasa es una alteración metabólica autosómica recesiva, que afecta la escisión de biotina disminuyendo su reciclado. Estudios en familiares del caso índice encontraron que generalmente ambos padres son portadores y los hermanos presentan el gen alterado; solo los homocigotos tienen síntomas que varían según el grado de deficiencia. Las madres pueden tener deficiencia moderada y mantenerse asintomáticas. En un estudio que utiliza células humanas expuestas a deficiencia de biotina, disminuyó el crecimiento celular y contribuyó al desarrollo de paladar hendido. La deficiencia de biotina en embarazadas ocasiona malformaciones en los productos. En recién nacidos, la deficiencia de biotinidasa se ha relacionado con síndrome VACTERL y páncreas anular. Se presenta el caso de una lactante con deficiencia de biotinidasa y defecto congénito de anillo vascular que rodea y comprime tráquea y esófago, alterando la deglución y la respiración. La niña fue suplementada con biotina e intervenida, con excelentes resultados.
Biotinidase deficiency is an autosomal recessive metabolic disorder that affects the cleavage of biotin. Family studies of the index case found that both parents are usually carriers and siblings have the altered gene, but only homozygotes have manifestations that vary depending on the deficiency grade. Mothers may have moderate deficiency and be asymptomatic; biotin deficiency in pregnant women causes defects in children. In a study, using human cells exposed to biotin deficiency, cell growth decreased contributing to the development of cleft palate. In newborns, biotinidase deficiency has been associated with VACTERL syndrome and annular pancreas. The case of an infant with biotinidase deficiency and congenital defect of the vascular ring is presented. This defect surrounds and compresses the trachea and esophagus, disturbing swallowing and breathing. Infant was supplemented with biotin and surgically intervened with excellent results.
Assuntos
Feminino , Humanos , Lactente , Aorta Torácica/anormalidades , Deficiência de Biotinidase/complicações , Artéria Subclávia/anormalidades , EsôfagoRESUMO
La deficiencia de biotinidasa es una alteración metabólica autosómica recesiva, que afecta la escisión de biotina disminuyendo su reciclado. Estudios en familiares del caso índice encontraron que generalmente ambos padres son portadores y los hermanos presentan el gen alterado; solo los homocigotos tienen síntomas que varían según el grado de deficiencia. Las madres pueden tener deficiencia moderada y mantenerse asintomáticas. En un estudio que utiliza células humanas expuestas a deficiencia de biotina, disminuyó el crecimiento celular y contribuyó al desarrollo de paladar hendido. La deficiencia de biotina en embarazadas ocasiona malformaciones en los productos. En recién nacidos, la deficiencia de biotinidasa se ha relacionado con síndrome VACTERL y páncreas anular. Se presenta el caso de una lactante con deficiencia de biotinidasa y defecto congénito de anillo vascular que rodea y comprime tráquea y esófago, alterando la deglución y la respiración. La niña fue suplementada con biotina e intervenida, con excelentes resultados.(AU)
Biotinidase deficiency is an autosomal recessive metabolic disorder that affects the cleavage of biotin. Family studies of the index case found that both parents are usually carriers and siblings have the altered gene, but only homozygotes have manifestations that vary depending on the deficiency grade. Mothers may have moderate deficiency and be asymptomatic; biotin deficiency in pregnant women causes defects in children. In a study, using human cells exposed to biotin deficiency, cell growth decreased contributing to the development of cleft palate. In newborns, biotinidase deficiency has been associated with VACTERL syndrome and annular pancreas. The case of an infant with biotinidase deficiency and congenital defect of the vascular ring is presented. This defect surrounds and compresses the trachea and esophagus, disturbing swallowing and breathing. Infant was supplemented with biotin and surgically intervened with excellent results.(AU)
RESUMO
Biotinidase deficiency is an autosomal recessive metabolic disorder that affects the cleavage of biotin. Family studies of the index case found that both parents are usually carriers and siblings have the altered gene, but only homozygotes have manifestations that vary depending on the deficiency grade. Mothers may have moderate deficiency and be asymptomatic; biotin deficiency in pregnant women causes defects in children. In a study, using human cells exposed to biotin deficiency, cell growth decreased contributing to the development of cleft palate. In newborns, biotinidase deficiency has been associated with VACTERL syndrome and annular pancreas. The case of an infant with biotinidase deficiency and congenital defect of the vascular ring is presented. This defect surrounds and compresses the trachea and esophagus, disturbing swallowing and breathing. Infant was supplemented with biotin and surgically intervened with excellent results.
Assuntos
Aorta Torácica/anormalidades , Deficiência de Biotinidase/complicações , Artéria Subclávia/anormalidades , Esôfago , Feminino , Humanos , LactenteRESUMO
Biotinidase deficiency is an autosomal recessive metabolic disorder that affects the cleavage of biotin. Family studies of the index case found that both parents are usually carriers and siblings have the altered gene, but only homozygotes have manifestations that vary depending on the deficiency grade. Mothers may have moderate deficiency and be asymptomatic; biotin deficiency in pregnant women causes defects in children. In a study, using human cells exposed to biotin deficiency, cell growth decreased contributing to the development of cleft palate. In newborns, biotinidase deficiency has been associated with VACTERL syndrome and annular pancreas. The case of an infant with biotinidase deficiency and congenital defect of the vascular ring is presented. This defect surrounds and compresses the trachea and esophagus, disturbing swallowing and breathing. Infant was supplemented with biotin and surgically intervened with excellent results.