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Abstract Aim: This study aimed to explore the effects of a training program during the pre-season on aerobic and anaerobic performance, hemodynamics, autonomic variables, and sleep quality in youth soccer players. Methods: Nineteen athletes, with an average age of 17 ± 1 years, participated in the study. The multicomponent training protocol was divided into technical, tactical, and physical practice for four weeks. The cardiac autonomic modulation was obtained through an electrocardiogram and blood pressure values were measured by a sphygmomanometer. The athletes answered the Pittsburgh questionnaire that assessed sleep patterns and issues. The VO2max was analyzed using the Intermittent Recovery Test Yo-Yo level 1. The RAST test was used to assess anaerobic power. Results: There was improvement in Heart Rate Variability (HRV) indicated by the increase in indexes, mean square root of the differences between normal cycles (RMSSD), low frequency increase (LF) (p = 0.04; d = 0.70), high frequency decrease (HF) (p = 0.01; d = 1.02) and the LF / HF sympathovagal index (p = 0.03; d = 0.70), variables related to faster recovery. An improvement in the components of sleep duration (p = 0.03) and quality (p = 0.02) of baseline and post-intervention sleep was also observed. Conclusion: The four-week multicomponent protocol contributed to improving VO2max, improving fatigue rates, quality of sleep, and maximum power. Additionally, we observe that youth soccer athletes had physiological and hemodynamic adaptations that resulted in an improvement in cardiac autonomic modulation and sleep patterns after four weeks of training.
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Humanos , Futebol/fisiologia , Estresse Fisiológico/fisiologia , Atletas , Qualidade do SonoRESUMO
BACKGROUND: Pulmonary Arterial Hypertension (PAH) is a severe and progressive disease of pulmonary arterioles. This pathology is characterized by elevation of the pulmonary vascular resistance and pulmonary arterial pressure, leading to right heart failure and death. Studies have demonstrated that resveratrol possesses a protective effect on the mechanisms related to the genesis of the PAH-induced by different models. OBJECTIVE: This study aimed to investigate the dose-related effects of resveratrol in different models of pulmonary arterial hypertension. METHODS: To identify eligible papers, we performed a systematic literature search on Scielo, Pub- Med, and Scholar Google. The research was limited to articles written in English in the last 10 years. We used the following descriptors to search: Pulmonary Arterial Hypertension and Resveratrol, OR Resveratrol, and Animal models of Pulmonary Arterial Hypertension, OR Resveratrol, and in vitro models of Pulmonary Arterial Hypertension. RESULTS: 1724 studies were identified through the descriptors used, fifty-five studies with different models of pulmonary arterial hypertension were selected for the full review, forty-four were excluded after application of exclusion and inclusion criteria, totalizing eleven studies included in this systematic review. CONCLUSION: The results showed that resveratrol, at low and high doses, protects in a dosedependent manner against the development of PAH induced through monocrotaline, normoxia and hypoxia models. In addition to having chemopreventive, anti-inflammatory, antioxidant and antiproliferative properties. In the case of PAH-related myocardial injury, resveratrol protects cells from apoptosis, thus working as an antiapoptotic agent.
Assuntos
Antioxidantes/uso terapêutico , Hipertensão Arterial Pulmonar/tratamento farmacológico , Resveratrol/uso terapêutico , Animais , Antioxidantes/farmacologia , Modelos Animais de Doenças , Humanos , Masculino , Camundongos , Resveratrol/farmacologiaRESUMO
BACKGROUND: Breast cancer is one of the leading causes of death worldwide. Heart rate variability (HRV) has attracted scientific community attention in different pathologies, becoming thus an ultimate importance tool in both clinical and research setting, being a good predictor of cardiac events and mortality risk and also used in physical exercise and sports in general. The aim of the present study was to evaluate 12 weeks of exercise training and six weeks of detraining in cardiorespiratory capacity, and autonomic modulation in breast cancer patients. METHODS: The sample was composed of 18 females (9 controls and 9 exercised), (aged 30-60 years). The HRV in the time and frequency domain was performed using an electrocardiogram before, after 12 weeks of the session of exercise training and after six weeks of detraining. Shapiro-Wilk and Mann-Whitney tests were made. RESULTS: No significant changes in time domain were found. In the frequency domain, 12 weeks of exercise training promote a decrease in LF (nu) and decrease in HF (nu) Index. The exercise training period promoted a decrease in LF/HF. The autonomic data returned to baseline levels after the detraining period. However, cardiorespiratory capacity remained increased after the detraining period. CONCLUSIONS: These data demonstrated that exercise training can be used to prevent autonomic dysfunction in breast cancer patients, but detraining promotes loss of all autonomic benefits.