RESUMO
The authors made a retrospective analysis of 136 cases of premature rupture of membranes (RPM) to check for the presence of neonatal infection, anoxia, and prematurity according to the latency period between rupture of membranes and parturition. Neonatal infection due to RPM has been the cause of 8.8% of clinically and laboratorily confirmed cases; in 6.6% of the cases, laboratorial confirmation was not possible. Neonatal infection was not influenced by the latency period between RPM and parturition. Maternal symptoms of infection were more frequent in the group with latency period longer than 24 hours and there was a significant relationship between maternal infection symptoms and neonatal symptoms. Prematurity and anoxia were detected in 20% of the cases, but no relation to RPM was observed.
Assuntos
Infecções Bacterianas/etiologia , Ruptura Prematura de Membranas Fetais/complicações , Doenças do Prematuro/etiologia , Líquido Amniótico/microbiologia , Infecções Bacterianas/diagnóstico , Feminino , Idade Gestacional , Humanos , Recém-Nascido , Gravidez , Estudos Retrospectivos , Fatores de TempoRESUMO
Progressive antighrombin activity was assayed in 45 term infants in the first four days of life, in 20 cord blood specimens and in the blood of 20 pregnant women (at term). In the term infants a positive correlation was shown between progressive antithrombin and postnatal age. No correlation could be demonstrated with sex, ethnic group or type of delivery. Maternal antithrombin activity was shown to be moderately decreased, maternal values being closely related to cord blood values.