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PURPOSE OF REVIEW: Posterior shoulder instability is an uncommon but important cause of shoulder dysfunction and pain which may occur as the result of seizure, high energy trauma, or repetitive stress related to occupational or sport-specific activities. This current review details the imaging approach to the patient with posterior shoulder instability and describes commonly associated soft tissue and bony pathologies identified by radiographs, CT, and MR imaging. RECENT FINDINGS: Advances in MR imaging technology and techniques allow for more accurate evaluation of bone and soft tissue pathology associated with posterior shoulder instability while sparing patients exposure to radiation. Imaging can contribute significantly to the clinical management of patients with posterior shoulder instability by demonstrating the extent of associated injuries and identifying predisposing anatomic conditions. Radiologic evaluation should be guided by clinical history and physical examination, beginning with radiographs followed by CT and/or MRI for assessment of osseous and soft tissue pathology. Synthesis of a patient's clinical history, physical exam findings, and radiologic examinations should guide clinical management.
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Introduction: Posterior glenohumeral joint dislocations are uncommon injuries. The disease often goes undiagnosed and untreated despite positive clinical signs. In the event of a large humeral bone defect, the outcome may be worse. Case Report: This case report presents a 47-year-old man with a 2-year-old locked posterior shoulder dislocation with a large segmental bone defect involving 40% of the humeral head's articular surface. We decided to treat the patient with an open reduction of the shoulder dislocation and reconstruction of the articular surface with iliac crest autograft. We observed an improvement in shoulder mobility and range of motion on all planes, which were beneficial to the patient's daily activities; at follow-up, no pain was reported. Conclusion: Our case report demonstrates that locked posterior shoulder dislocations with a large bone defect and viable humeral head can be treated using an autograft. This will result in optimal clinical results and avoid early prosthetic replacement surgery. Autograft may be a preferred method in the presence of economic constraints or due to the unavailability of allografts or implants.
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BACKGROUND: The primary aim of this study was to investigate bone loss in the glenoid with magnetic resonance imaging in posterior shoulder instability with only a labral tear. METHODS: A total of 76 patients operated on because of posterior and anteroposterior shoulder instability only with a labral tear between 2006 and 2019 (n = 40 and n = 36, respectively) were included in this study. The instability type, a presence of an additional superior labrum anteroposterior (SLAP) lesion, the number of dislocations, and the magnetic resonance imaging-based measurements (the glenoid diameter and the bone defect size in the glenoid, the Hill-Sachs lesion [HSL] and the reverse HSL [rHSL] length, the angle and the arc length of HSL and rHSL, and the humerus head diameter and its area) were analyzed. RESULTS: The size of the anterior glenoid defect, the rHSL measurements (length, angle, and arc length), and the ratio of the anterior glenoid defect size to the glenoid diameter were significantly higher for anteroposterior instability (P < .01) cases. There was no significant difference (P = .49, .64, and .82, respectively) for the presence of an additional SLAP pathology, the glenoid diameter, the posterior glenoid defect, and the ratio of the posterior glenoid defect size to the glenoid diameter in posterior and anteroposterior instability groups. The increased number of dislocations was associated with increased rHSL length and total arc length (P = .04 and .03, respectively). An additional SLAP lesion in posterior shoulder instabilities was not associated with the bone defect size (P = .29). CONCLUSION: Although the posterior shoulder instability with only a labral tear is likely to cause a bone defect, we have shown that the instability is not expected to be caused by the bone defect. Therefore, this study points out that only soft tissue repair without considering the bone defect could be promising in this patient group.
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Lesiones de Bankart , Enfermedades Óseas Metabólicas , Luxaciones Articulares , Inestabilidad de la Articulación , Laceraciones , Luxación del Hombro , Articulación del Hombro , Humanos , Articulación del Hombro/diagnóstico por imagen , Articulación del Hombro/cirugía , Articulación del Hombro/patología , Luxación del Hombro/complicaciones , Luxación del Hombro/diagnóstico por imagen , Luxación del Hombro/cirugía , Hombro/patología , Inestabilidad de la Articulación/diagnóstico por imagen , Inestabilidad de la Articulación/cirugía , Inestabilidad de la Articulación/etiología , Rotura/complicaciones , Imagen por Resonancia Magnética/métodos , Luxaciones Articulares/complicaciones , Lesiones de Bankart/patología , RecurrenciaRESUMEN
Historically, posterior shoulder instability has been a challenging problem for contact athletes and orthopedic surgeons alike. A complete understanding of the normal shoulder anatomy and biomechanics and the pathoanatomy responsible for the instability is necessary for a successful clinical outcome. In addition, the surgeon must be familiar with the diagnostic imaging and physical examination maneuvers required for the correct diagnosis without missing any other concurrent abnormalities. This understanding will allow orthopedists to plan and execute the appropriate management, whether this may involve conservative or surgical intervention. The goal should always be to correct the abnormality and have the patient return to play with full strength and no recurrent instability.