RESUMEN
A part of patients' arterial hyperglycemia is not accompanied by increases in the content of glucose in brain substance, and aggressive correction of hyperglycemia can lead to reduction in concentration of glucose expressed in "affected", and conditionally "intact" brain substance. At choosing a mixed strategy of parenteral-enteral correction should take into account not only condition of the gastrointestinal tract but especially absorption of glucose (the level of cellular homeostasis).
Asunto(s)
Encéfalo/metabolismo , Nutrición Enteral/métodos , Tracto Gastrointestinal/metabolismo , Glucosa/metabolismo , Hiperglucemia/metabolismo , Hemorragias Intracraneales/terapia , Nutrición Parenteral/métodos , Adolescente , Glucemia/análisis , Metabolismo Energético , Tracto Gastrointestinal/fisiopatología , Humanos , Hiperglucemia/prevención & control , Hemorragias Intracraneales/sangre , Hemorragias Intracraneales/metabolismo , Hemorragias Intracraneales/fisiopatología , Masculino , Úlcera Péptica/prevención & control , Índice de Severidad de la EnfermedadRESUMEN
Thirty-eight patients with mushroom (Paxillus involutus and Paxillus atrotomentosus) poisoning were treated. Slight poisoning (acute gastroenteritis) was diagnosed in 17 patients, medium-severe in 13, severe in 6, and extremely severe in 2 patients. Changes in the LPO-AOD system correlated with the severity of hepatorenal involvement. The treatment included hepatotropic therapy; patients with acute renal failure were treated by hemodialysis. Paxillus mushrooms induced functional evacuatory disorders in the small intestine. Eleven patients with adhesions in the abdominal cavity developed ileus. Two patients died: a man aged 26 years after eating fried (not boiled) mushrooms and a woman aged 76 years with ileus with symptoms of multiple organ dysfunction. The rest patients were discharged from hospital in satisfactory condition. Clinical course of poisoning with Paxillus mushrooms is discussed.