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INTRODUCTION: Adolescent health risk behaviors are linked to poor physical and mental health outcomes. While past research shows that maternal psychological distress predicts those behaviors, we know less about the role of paternal psychological distress and the role of sensitive periods. METHODS: Using 11,128 data from families (50.5% female children) from the UK's Millennium Cohort Study, we examined the role of timing of exposure to paternal and maternal psychological distress in engagement in health risk behaviors (smoking, alcohol use, binge drinking, and sexual activity) at age 14. Paternal and maternal psychological distress, measured with the Kessler-6 scale, were assessed at child ages 3, 7, and 11. We performed path analysis, adjusting for key covariates, modeling maternal distress parallel to paternal, and allowing for autoregressive paths. RESULTS: Paternal distress experienced at age 11 predicted a higher likelihood of smoking at age 14. Maternal distress at age 7 also predicted a higher likelihood of smoking, alcohol use, and binge drinking, but only for boys. Moreover, maternal distress at age 3 was associated with a lower risk for alcohol use. Effects were not replicated in the sensitivity analysis we performed, including only families with resident biological fathers across the study period. Instead, maternal and paternal distress at age 11 raised girls' risk for binge drinking and sexual activity, respectively. CONCLUSIONS: Parental distress in early childhood does not predict adolescent health risk behaviors. In late childhood, however, both paternal and maternal distress seem to influence the likelihood of engagement in such behaviors.
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Household chaos has been shown to adversely associate with children's behavioral adjustment. However, the mechanism underlying the relationship between household chaos and children's behaviors is not yet fully understood. The current study proposes mindful parenting as an important mediating and moderating factor in the relationship between household chaos and child problem behaviors. This study also examines cultural influences in this process, comparing the UK and Türkiye, considering both mothers' and children's perspectives on mindful parenting. Cross-sectional questionnaires were administered to mothers and their children aged 11-16 years in the UK (n = 90; 53.3% girl) and Türkiye (n = 154; 54.5% girl) in 2021. Mother reports of the Confusion, Hubbub, and Order Scale and the Strengths and Difficulties Questionnaire, as well as mother and child reports of Mindful Parenting Inventories for Parents (MPIP) and Children (MPIC), were used to assess household chaos, child problem behaviors, and mindful parenting, respectively. Multiple-group path analysis revealed that household chaos was a significant indirect predictor of child problem behaviors via mindful parenting in both countries. Furthermore, simple slopes analysis showed that mindful parenting moderated the link between household chaos and child problem behaviors in the UK. Overall, our study sheds light on the importance of micro- and macro-environmental factors and their interactions in children's adjustment.
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Comparación Transcultural , Responsabilidad Parental , Humanos , Femenino , Responsabilidad Parental/psicología , Niño , Masculino , Adolescente , Estudios Transversales , Reino Unido , Encuestas y Cuestionarios , Problema de Conducta/psicología , Atención Plena , Conducta Infantil/psicología , Madres/psicología , Composición Familiar , AdultoRESUMEN
Autistic children show higher rates of co-occurring emotional and behavioural difficulties compared to other children in the general population. However, the environmental factors which contribute to the development of emotional (internalising) and behavioural (externalising) difficulties in autistic individuals are poorly understood. This study sought to investigate the association between harsh parenting (smacking, shouting, telling off) and the trajectories of emotional and behavioural difficulties in autistic children from ages 3 to 7. A sample comprising of 349 autistic children participated from the UK's Millennium Cohort Study. Associations between harsh parenting and child emotional and behavioural difficulties were modelled using multilevel growth curve models. In autistic children, harsh parenting was associated with total emotional and behavioural difficulties and behavioural (but not emotional) problems concurrently. Moreover, harsh parenting was not associated with changes in emotional and behavioural difficulties over time. Harsh parenting may have an important role in externalising problems in young autistic children indicating the need for public health strategies which educate parents on its effects.
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Research over many decades has considered the crucial role of maternal psychological distress (e.g., depression, anxiety) for children's psychological adjustment (externalizing and internalizing problems), suggesting bidirectional influences over time. However, little is known about the extent to which household chaos (e.g., noise, disorganization, lack of calm) may mediate this mutual association, despite an understanding that chaos is a powerful stressor in the home. Conducting secondary data analysis in a large scale, prospective longitudinal study of families with children-the U.K.'s Millennium Cohort Study-we accounted for stability in both maternal psychological distress and children's internalizing and externalizing problems and examined the extent to which household chaos mediated the mutual association between maternal psychological distress and children's behaviors. Using what we term a mutual-mediation model, we found that both maternal psychological distress and children's adjustment problems predicted household chaos at Age 5, and in turn, that chaos predicted maternal psychological distress and child adjustment problems at Age 7. We found a dominance of children's externalizing problems in the prediction of household chaos, and all pathways were strongest for maternal psychological distress and externalizing problems compared to child internalizing problems. Our findings suggest that research would be well-minded to consider both child and parent effects on household chaos, as well as its mediation potential. (PsycInfo Database Record (c) 2023 APA, all rights reserved).
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Familia , Distrés Psicológico , Niño , Humanos , Preescolar , Femenino , Estudios Longitudinales , Estudios de Cohortes , Estudios Prospectivos , Familia/psicología , Madres/psicologíaRESUMEN
OBJECTIVE: Parental psychological distress (depression, anxiety) is detrimental to child mental health. A key reason for this is that depressed and anxious parents are at risk of engaging in more negative, reactive and harsh parenting. While treatment for psychological distress has a long history of success in adults, less is known about how treatment for parental psychological distress may positively influence parenting behaviours, particularly in the general population. We examined the moderating role of mothers receiving treatment for depression or anxiety on the longitudinal relationship between maternal psychological distress and the development of harsh parenting (smacking and shouting) across early childhood (ages 3 to 7). METHOD: Using prospective data from 16,131 families participating in the UK's Millennium Cohort Study, we conducted moderator analysis within a multilevel repeated measures model to test whether receiving treatment for mental health problems could protect mothers with high psychological distress from engaging in harsh parenting. RESULTS: In each wave, about 7% of mothers reported undergoing treatment for depression or anxiety at that time. Maternal psychological distress was associated with increased use of harsh parenting and that, adjusting for psychological distress, receiving psychological treatment was related to decreased use of harsh parenting. Importantly, receiving psychological treatment buffered the negative effect of psychological distress on harsh parenting. CONCLUSION: In early-to-middle childhood, mental health treatment may help mothers with depression or anxiety to be less harsh toward their children, thereby benefiting their child's psychological adjustment.
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Madres , Responsabilidad Parental , Femenino , Adulto , Humanos , Niño , Preescolar , Responsabilidad Parental/psicología , Estudios Prospectivos , Estudios de Cohortes , Madres/psicología , Ansiedad/psicología , Estrés Psicológico/psicologíaRESUMEN
BACKGROUND: Autism can be diagnosed from 2 years of age, although most autistic people receive their diagnosis later than this after they have started education. Research is required to understand why some autistic children are diagnosed late, and the level and nature of unmet need prior to diagnosis for late-diagnosed children. METHODS: We examined trajectories of emotional, behavioural and social difficulties (EBSDs) across childhood and adolescence, comparing 'earlier-diagnosed' (diagnosed 7 years or younger) with 'late-diagnosed' (diagnosed between 8 and 14 years) autistic children. Data were from the Millennium Cohort Study, a population-based UK birth cohort. EBSDs were measured using the parent-report Strengths and Difficulties Questionnaire, at 3, 5, 7, 11 and 14 years. We used Growth Curve Modelling to investigate levels and rates of change in these difficulties, and to compare earlier- (n = 146) and late-diagnosed (n = 284) autistic children. RESULTS: Aged 5, earlier-diagnosed autistic children had more emotional (i.e., internalising), conduct, hyperactivity and social difficulties; although clinical difficulties in these areas were nevertheless common in late-diagnosed children. There was a faster annual increase in scores for all domains for late-diagnosed children, and by age 14 years, they had higher levels of EBSDs. These results persisted when we ran adjusted models, to account for the late-diagnosed group having higher rates of late-diagnosed attention deficit/hyperactivity disorder, higher IQ, a higher proportion of females and older and more educated mothers. CONCLUSIONS: Emotional, behavioural and social difficulties are associated with, and may influence, the timing of autism diagnosis. Late-diagnosed autistic children often have high levels of mental health and social difficulties prior to their autism diagnosis, and tend to develop even more severe problems as they enter adolescence.
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Trastorno por Déficit de Atención con Hiperactividad , Trastorno Autístico , Femenino , Niño , Adolescente , Humanos , Trastorno Autístico/diagnóstico , Trastorno Autístico/psicología , Estudios de Cohortes , Salud Mental , EscolaridadRESUMEN
Ability-grouping has been studied extensively in relation to children's academic, but not emotional and behavioral outcomes. The sample comprised 7259 U.K. children (50% male) with data on between-class and within-class ability-grouping at age 7. Peer, emotional, hyperactivity, and conduct problems were measured at ages 7, 11, and 14 years. Children in low within-class ability groups showed more hyperactivity and emotional problems across the study period compared to non-grouped children, after adjustments for the different types of ability grouping and confounding. Additionally, children in the middle within-class ability groups showed more, and those in the top within-class groups less, hyperactivity compared to non-grouped children, after adjustment. Children in lower within-class groups should be monitored closely to ensure that their well-being is not compromised.
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Problema de Conducta , Adolescente , Niño , Emociones , Femenino , Humanos , Masculino , Grupo Paritario , Problema de Conducta/psicologíaRESUMEN
High rates of psychological distress including anxiety and depression are common in the doctoral community and the learning environment has a role to play. With the coronavirus disease (Covid-19) pandemic taking a toll on mental health it is necessary to explore the risk and protective factors for this population. Using data from the Covid-19: Global Study of Social Trust and Mental Health, the present study examined the relationship between Covid-19-related stressful educational experiences and doctoral students' mental health problems. Moreover, it assessed the role of attentional ability and coping skills in promoting good mental health. One hundred and fifty-five doctoral students completed an online survey where micro-, meso- and macro-level educational stressors were measured. The Patient Health Questionnaire and the Generalized Anxiety Disorder Questionnaire were used to measure depression and anxiety symptoms, respectively. We also measured coping skills using a 13-item scale and attentional ability using a questionnaire. The results of multiple linear regression analyses showed that specific stressful educational experiences were unrelated but cumulative stressful educational experiences were related to increased depression symptoms (but not anxiety symptoms) in fully adjusted models. Additionally, higher coping skills and attentional ability were related to fewer depression and anxiety symptoms. Finally, no associations between demographics and other covariates and mental health problems were found. The experience of multiple educational stressful events in their learning environment due to Covid-19 is a key risk factor for increased mental illness in the doctoral community. This could be explained by the uncertainty that the Covid-19 pandemic has caused to the students.
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Research shows that paternal psychological distress is associated with child emotional and behavioural difficulties. However, little is known about the direction of this association including whether it is bidirectional. The aim of this study was to explore the reciprocal relationships between paternal psychological distress and child emotional and behavioural problems longitudinally (at ages 3, 5, 7, 11 and 14 years) in a sample of 13,105 children (49% girls) who participated in the UK's Millennium Cohort Study (MCS), a large-scale, nationally representative, longitudinal survey. Four domains of child problems (emotional symptoms, peer relations, conduct problems and hyperactivity/inattention) were measured with the Strengths and Difficulties Questionnaire and paternal psychological distress was measured with the Kessler K-6 scale. Data were analysed using cross-lagged path models. At all ages, paternal psychological distress predicted both subsequent child emotional symptoms as well as peer problems. Paternal psychological distress at child's age 3 was related to more hyperactivity at age 5 and, at age 5, paternal psychological distress was associated with more conduct problems at age 7. At age 11, paternal distress was also related to age 14 conduct problems and hyperactivity. Child effects were fewer and were found mainly for behavioural problems. Notably, we found bidirectional links between paternal psychological distress and child peer difficulties, from 11 to 14 years. Paternal psychological distress appears to influence child behaviour more consistently than the converse. However, in early adolescence, there appears to be a reciprocal relationship between fathers' mental health problems and children's peer problems.
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Trastornos de la Conducta Infantil , Padre , Distrés Psicológico , Adolescente , Niño , Trastornos de la Conducta Infantil/epidemiología , Preescolar , Estudios de Cohortes , Padre/psicología , Femenino , Humanos , MasculinoRESUMEN
BACKGROUND: Family financial difficulties have been directly linked to poorer executive functioning in childhood. However, recent studies suggest that difficulties in affording basic items and other necessities may also indirectly affect children's executive functions through several psychological but also physiological paths. One of the latter may be inflammation, which has been related to both financial difficulties and executive functioning. In this study, we explored for the first time if the relationship between early family financial difficulties and working memory in middle childhood can be explained by inflammation. METHODS: Using data from 4,525 children of the Avon Longitudinal Study of Parents and Children, a general population birth cohort, we tested associations between parents' perceptions at ages 0-3 years of having difficulties in affording basic items for their children including food and clothing, children's inflammation [measured by interleukin 6 (IL-6) and C-reactive protein (CRP)] at age 9 years and working memory performance at age 10 years. Confounders included socioeconomic status at ages 0-3 years, economic hardship between ages 3-9 years, BMI and gender. RESULTS: Using Structural Equation Modelling, we found that financial difficulties were associated with worse working memory (ß=-0.076, 95 % CI=-0.105, -0.043) even after adjusting for confounders. This association was partially explained by inflammation (ß=-0.002, 95 % CI=-0.005, -0.001) as measured by IL-6. CONCLUSIONS: Children in families struggling to afford necessities early in life have higher levels of inflammation, in turn related to poorer executive functioning in middle childhood. These findings suggest that living under financial strain has a unique effect on children's cognitive development through inflammation in the general population.
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Inflamación , Memoria a Corto Plazo , Pobreza , Cohorte de Nacimiento , Proteína C-Reactiva/análisis , Niño , Preescolar , Femenino , Humanos , Lactante , Recién Nacido , Inflamación/sangre , Inflamación/fisiopatología , Interleucina-6/sangre , Estudios Longitudinales , Masculino , Memoria a Corto Plazo/fisiologíaRESUMEN
Psychotic-like experiences (PLEs) are part of a continuum of psychosis. Previous longitudinal studies highlighted a relationship between peripheral inflammation during childhood and onset of PLEs in adulthood. In this study, we tested if this association is mediated by internalising and externalising symptoms experienced during childhood and adolescence. To test this hypothesis, we used data from the Avon Longitudinal Study of Parents and Children (ALSPAC). We investigated a subsample of 4525 individuals from this cohort with data on interleukin 6 (IL-6) and C-reactive protein (CRP) in childhood (age 9â¯years). We measured PLEs at age 18â¯years, and we used latent growth curve modelling to estimate longitudinal trajectories of internalising and externalising symptoms from ages 9 to 16â¯years. The individual predicted values of the intercept (set at baseline, 9â¯years) and the slope (rate of annual change) were then used in the mediation analysis. There was evidence for full mediation by the intercept of internalising symptoms. Our findings suggest that inflammation during childhood may be relevant for the future onset of PLEs via its association with a high level of internalising symptoms. These findings, although obtained from a non-clinical population, provide an additional step in advancing knowledge on the relationship between inflammation and symptoms of the psychosis continuum.
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Inflamación , Trastornos Psicóticos , Adolescente , Adulto , Proteína C-Reactiva/análisis , Niño , Estudios de Cohortes , Humanos , Inflamación/epidemiología , Estudios Longitudinales , Trastornos Psicóticos/epidemiologíaRESUMEN
This study aimed to identify determinants of a late autism spectrum disorder diagnosis, including diagnoses made 'very late' (i.e., in adolescence), using the Millennium Cohort Study, a nationally representative population-based cohort in the United Kingdom. Children diagnosed with autism spectrum disorder by age 14 (N = 581) were included and grouped by the parent-reported timing of diagnosis: before school (up to age 5), during primary school (age 5-11) and during secondary school (age 11-14). Predictors of diagnostic timing, at the child, family and school levels, were investigated using multinomial logistic regression. Most (79%) children with autism spectrum disorder were diagnosed after school entry, and 28% were not diagnosed until secondary school. Among those not diagnosed until secondary school, 75% had been identified at age 5 years by a parent and/or teacher as having socio-behavioural difficulties. Being diagnosed after starting school was predicted by living in poverty (adjusted relative risk ratio: primary = 1.90, 95% confidence interval: 1.03-3.53; secondary = 2.15, 1.05-4.42) and/or having no initial parental concerns (primary = 0.32, 0.15-0.70; secondary = 0.19, 0.09-0.43). Having typical-range intelligence also predicted diagnosis during secondary school. The result indicates that those without cognitive delays and poorer children were at risk of 'very late' (i.e. adolescent) diagnosis. Strategies to promote earlier identification, targeting age at primary school entry, could help those more likely to be diagnosed late.Lay abstractDespite policy emphasis on early identification, many children with autism spectrum disorder are diagnosed late, with some being diagnosed as late as in adolescence. However, evidence on what determines the timing of autism spectrum disorder diagnosis including children diagnosed in adolescence is lacking. Understanding these determinants, particularly in those diagnosed later than is ideal, can inform the development of effective strategies to improve earlier identification of autism spectrum disorder. This study used a nationally representative population-based cohort in the United Kingdom to explore child, family and school level predictors of timing of autism spectrum disorder diagnosis. In the United Kingdom, 79% of the children with autism spectrum disorder were diagnosed after entering primary school and 28% during secondary school. Among those not diagnosed until secondary school, 75% had shown social difficulties noticed by parents and/or teachers at age 5 years. The results suggest that healthcare providers should be aware that, even for universal systems of care, those living in poverty and having higher intelligence are most likely to miss out on a timely diagnosis. Strategies to promote earlier identification among school-aged children, including targeting primary school entry age (i.e. around age 5) and that encouraging referrals for a formal assessment at the first report of concerns over the child's social development may benefit those children who would otherwise be diagnosed later.
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Trastorno del Espectro Autista , Adolescente , Trastorno del Espectro Autista/diagnóstico , Trastorno del Espectro Autista/epidemiología , Niño , Preescolar , Estudios de Cohortes , Humanos , Padres , Instituciones Académicas , Reino Unido/epidemiologíaRESUMEN
BACKGROUND: Stressful life events experienced during childhood and early prenatal development have been associated with inflammation during childhood. However, no study has considered these two exposures jointly, or has investigated the effect of their interaction. METHODS: In the Avon Longitudinal Study of Parents and Children, a general-population birth cohort, we explored if inflammatory markers [serum C-reactive protein (CRP) and interleukin 6 (IL-6)] at age 9 years were related to early prenatal events (at 18 weeks pregnancy), childhood events (measured on seven occasions at ages 0-9 years) and their interaction (n = 3,915). Latent growth curve modelling estimated trajectories of childhood events, and linear regression explored associations of prenatal and childhood events with inflammatory markers. Models controlled for ethnicity, socioeconomic status and body mass index, were stratified by gender and considered both unweighted and weighted (by impact) event exposures. RESULTS: Even after adjustment for confounders and prenatal events, both the intercept and the slope of number of childhood events were associated with IL-6, but only in females. The significant effect of the slope held for both weighted (by impact) and unweighted event specifications. Prenatal events were not associated with either inflammatory marker when childhood events were controlled. There was no evidence for synergistic effects of prenatal and childhood events. CONCLUSION: Independently of prenatal adverse life events, the number and increase in number of adverse life events experienced in childhood were associated positively with plasma levels of inflammatory markers, such as IL-6, in girls. This gender specificity warrants further research.
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Inflamación , Efectos Tardíos de la Exposición Prenatal , Biomarcadores , Proteína C-Reactiva/análisis , Niño , Preescolar , Femenino , Humanos , Lactante , Recién Nacido , Interleucina-6 , Estudios Longitudinales , EmbarazoRESUMEN
BACKGROUND: No study has investigated the role of inflammation in explaining the association between early exposures to adverse life events and depressive symptoms in adolescence. METHOD: Using data from the Avon Longitudinal Study of Parents and Children, we tested if inflammatory markers [serum C-reactive protein (CRP) and interleukin 6 (IL-6)] at age 9 years mediate the association between adverse life events, measured separately for the prenatal (since the beginning of pregnancy) and the childhood (ages 0-9 years) periods, and the development of depressive symptoms at ages 10-17 years. Data (nâ¯=â¯4,263) were analyzed using mediation analysis in a latent growth curve modeling framework. RESULTS: Depressive symptoms at the beginning of adolescence (age 10) were associated with the number of prenatal events, the number of events around birth and the increase in events over time in childhood (ages 0-9), even after adjustment for confounders. IL-6 partially mediated the association between increasing exposure to events over time in childhood and depressive symptoms at the beginning of adolescence. IL-6 did not mediate any other association between events and symptoms. There was no evidence for mediation by CRP, which was generally unrelated to exposure to events. LIMITATIONS: The small size of the mediation effect and the robust direct effects of events prenatally and around birth suggest there are multiple routes from early stressors to adolescent depression. CONCLUSIONS: In the general adolescent population, increasing exposure to psychosocial stressors over time during childhood is associated with the early onset of depressive symptoms, partly via increasing levels of plasma IL-6.
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Experiencias Adversas de la Infancia , Trastorno Depresivo/etiología , Inflamación/etiología , Efectos Tardíos de la Exposición Prenatal/etiología , Estrés Psicológico/etiología , Adolescente , Conducta del Adolescente/psicología , Biomarcadores/sangre , Proteína C-Reactiva/metabolismo , Niño , Preescolar , Trastorno Depresivo/sangre , Trastorno Depresivo/psicología , Femenino , Estudios de Seguimiento , Humanos , Lactante , Recién Nacido , Inflamación/sangre , Inflamación/psicología , Interleucina-6/sangre , Acontecimientos que Cambian la Vida , Masculino , Embarazo , Efectos Tardíos de la Exposición Prenatal/sangre , Efectos Tardíos de la Exposición Prenatal/psicología , Estudios Prospectivos , Estrés Psicológico/sangre , Estrés Psicológico/psicología , Encuestas y CuestionariosRESUMEN
BACKGROUND: Children from families of low socioeconomic status (SES) are more likely to be exposed to upsetting situations and stressors. Such exposures have, in turn, been linked to inflammation in some studies. In this study we explore if low SES is related to inflammation in children via such stressful life events. METHODS: Data on 4525 children of the Avon Longitudinal Study of Parents and Children, a general population birth cohort, were used to explore associations between SES at ages 0-3 years, upsetting life events at ages 3-9 years and inflammatory markers [interleukin 6 (IL-6) and C-reactive protein (CRP)] at age 9 years. Confounders included body mass index, gender, financial problems, and upsetting life events at ages 0-3 years. RESULTS: Using Structural Equation Modelling, we found that early socioeconomic disadvantage predicted higher levels of IL-6 (ß = 0.034, 95% CI = 0.005, 0.063) even after adjusting for confounders. This association was partially mediated by upsetting life events (ß = 0.003, 95% CI = 0.001, 0.006). CONCLUSIONS: In the general child population, low SES is associated with increased exposure to stressful life events, in turn associated with later inflammation. These findings highlight the role of stressors associated with poverty and disadvantage in the development of inflammation among children in the general population.
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Renta/estadística & datos numéricos , Inflamación/epidemiología , Clase Social , Proteína C-Reactiva/análisis , Niño , Preescolar , Femenino , Humanos , Recién Nacido , Inflamación/sangre , Interleucina-6/sangre , Estudios Longitudinales , MasculinoRESUMEN
Psychotic-like experiences (PLEs) are part of a continuum of psychosis. Previous longitudinal studies highlighted a relationship between peripheral inflammation during childhood and onset of PLEs in adulthood. In this study, we tested if this association is mediated by internalising and externalising symptoms experienced during childhood and adolescence. To test this hypothesis, we used data from the Avon Longitudinal Study of Parents and Children (ALSPAC). We investigated a subsample of 4525 individuals from this cohort with data on interleukin 6 (IL-6) and C-reactive protein (CRP) in childhood (age 9â¯years). We measured PLEs at age 18â¯years, and we used latent growth curve modelling to estimate longitudinal trajectories of internalising and externalising symptoms from ages 9 to 16â¯years. The individual predicted values of the intercept (set at baseline, 9â¯years) and the slope (rate of annual change) were then used in the mediation analysis. There was evidence for full mediation by the intercept of internalising symptoms. Our findings suggest that inflammation during childhood may be relevant for the future onset of PLEs via its association with a high level of internalising symptoms. These findings, although obtained from a non-clinical population, provide an additional step in advancing knowledge on the relationship between inflammation and symptoms of the psychosis continuum.
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Síntomas Conductuales/fisiopatología , Inflamación/sangre , Trastornos Psicóticos/fisiopatología , Adolescente , Síntomas Conductuales/epidemiología , Proteína C-Reactiva/metabolismo , Niño , Comorbilidad , Femenino , Humanos , Inflamación/epidemiología , Interleucina-6/sangre , Estudios Longitudinales , Masculino , Trastornos Psicóticos/epidemiología , Reino Unido/epidemiologíaRESUMEN
There is little research on the role of school and its composition in explaining individual children's psychological outcomes. This study examined for the first time the role of several primary-school compositional characteristics, and their interactions with individual level characteristics, in the development of two such outcomes, internalising and externalising problems, at ages 7, 11 and 14 years in 4794 children in England participating in the Millennium Cohort Study. Using hierarchical (multilevel) linear models, we found that, even after adjusting for individual and family characteristics, children in schools with higher proportions of pupils eligible for free school meals had more externalising problems. In general, children with special educational needs, lower academic performance, more distressed mothers, and those in non-intact families had more internalising and externalising problems. Our results underline the importance of targeting schools with less affluent overall intakes, but also highlight the key role of individual and family characteristics in the development of their pupils' psychological functioning.
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Éxito Académico , Síntomas Conductuales/epidemiología , Hijo de Padres Discapacitados/estadística & datos numéricos , Educación Especial/estadística & datos numéricos , Composición Familiar , Madres/estadística & datos numéricos , Pobreza/estadística & datos numéricos , Instituciones Académicas/estadística & datos numéricos , Adolescente , Niño , Estudios de Cohortes , Inglaterra/epidemiología , Femenino , Humanos , MasculinoRESUMEN
Inflammation has been proposed as a pathway from adverse physical environments to poor physical and mental health. We estimated longitudinal associations of neighbourhood-level air pollution and greenspace with individual-level inflammation (measured with C-reactive protein and fibrinogen), using data from over 8000 adults living in England and Wales who participated in Understanding Society. Using linear regression, we found that neighbourhood-level nitrogen dioxide predicted later levels of fibrinogen, but not C-reactive protein. Area air pollution, but not area greenery, appears to predict inflammation, even after accounting for social deprivation in the area.
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Contaminación del Aire/estadística & datos numéricos , Inflamación/epidemiología , Características de la Residencia , Adulto , Anciano , Anciano de 80 o más Años , Proteína C-Reactiva/análisis , Inglaterra/epidemiología , Femenino , Fibrinógeno/análisis , Humanos , Estudios Longitudinales , Masculino , Persona de Mediana Edad , Dióxido de Nitrógeno/análisis , Factores de Riesgo , Gales/epidemiologíaRESUMEN
BACKGROUND: This study explored the role of outdoor air pollution [nitrogen dioxide (NO2) and sulphur dioxide (SO2)] and indoor air quality (measured with damp or condensation and secondhand smoke exposures) at age 9 months in emotional, conduct and hyperactivity problems at age 3 years. METHOD: Data from 11,625 Millennium Cohort Study children living in England and Wales were modelled using multilevel regression. RESULTS: After adjusting for a host of confounders, having a damp or condensation problem at home was related to both emotional and conduct problems. Secondhand smoke exposure was associated with all three problem types. Associations with outdoor air pollution were less consistent. CONCLUSIONS: Exposures to damp or condensation and secondhand smoke in the home are likely to be risk factors for child emotional and behavioural problems. Parents should continue to be educated about the dangers of exposing their children to poor air quality at home.
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Contaminantes Atmosféricos/efectos adversos , Contaminación del Aire Interior/efectos adversos , Contaminación del Aire/efectos adversos , Trastornos de la Conducta Infantil/etiología , Niño , Conducta Infantil , Protección a la Infancia/estadística & datos numéricos , Preescolar , Estudios de Cohortes , Inglaterra , Femenino , Humanos , Lactante , Masculino , Dióxido de Nitrógeno/efectos adversos , Factores de Riesgo , Dióxido de Azufre/efectos adversos , Contaminación por Humo de Tabaco/efectos adversos , GalesRESUMEN
Callous-unemotional (CU) traits and male gender are both known risk factors for poor academic outcomes in children and adolescents. However, despite gender differences in CU trait severity, comorbid difficulties and correlates of CU traits, research has yet to examine whether the CU traits and male gender may work together to increase risk for poor academic performance. That is, whether boys high in CU traits perform more poorly across academic disciplines than girls high in these traits. This study therefore aimed to investigate i) the relationships between CU traits, student gender and English, Science and Math grades, and ii) whether gender moderates the association between CU traits and academic outcomes. Participants were 437 children aged 11 to 14 years (mean age 12.50 years; 49% girls; 85% White) attending a state secondary school in England. Students reported on CU traits and externalizing problems and their English, Math and Science grades were gathered from school records. Using hierarchical linear modelling, CU traits were found to be significantly related to lower English, Math and Science grades when controlling for age, gender, sociodemographic disadvantage and externalizing problems. CU traits were significantly related to lower Science grades for boys but not girls. However, gender did not moderate the association between CU traits for English or Math grades. Findings enhance our understanding of how child characteristics may interact to increase the likelihood of poor school outcomes, and therefore help us to identify youth at-risk for poor academic performance.