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Eur J Endocrinol ; 165(1): 129-36, 2011 Jul.
Artículo en Inglés | MEDLINE | ID: mdl-21551166

RESUMEN

OBJECTIVE: Autoimmune thyroid disease (AITD) is characterized by different defects in immunoregulatory mechanisms. The immunoglobulin-like transcript receptor 2 (ILT2) or leukocyte Ig-like receptor 1 (LIRB1/CD85j) exerts an important immunoregulatory role. We hypothesized that the lymphocytes from AITD patients have a diminished expression and function of ILT2. The aim of this study was to investigate the expression and function of ILT2 in lymphocytes from patients with AITD. DESIGN AND METHODS: In this study, 18 patients with Hashimoto's thyroiditis (HT), 20 with Graves' disease, and 26 healthy controls were studied. ILT2 expression was analyzed by flow cytometry and immunohistochemistry in peripheral blood mononuclear cells (PBMC) and thyroid tissue. The regulatory function of ILT2 was assessed by an assay of inhibition of lymphocyte proliferation and by an analysis of cell cycle progression. The effect of ILT2 on cytokine synthesis was also evaluated. RESULTS: We found a significant increased expression of ILT2 by lymphocytes in AITD patients. ILT2 was also detected in the leukocyte infiltrate of thyroid tissue from HT patients. On the contrary, a significant diminished inhibitory activity of ILT2 on cell proliferation was observed in AITD patients. In addition, PBMC from AITD patients showed a diminished synthesis of interleukin 10 on ILT2 engagement. CONCLUSIONS: The abnormal expression and function of ILT2 detected in AITD suggests that this receptor may participate in the pathogenesis of this condition.


Asunto(s)
Antígenos CD/biosíntesis , Antígenos CD/fisiología , Enfermedad de Graves/inmunología , Enfermedad de Hashimoto/inmunología , Linfocitos/inmunología , Receptores Inmunológicos/biosíntesis , Receptores Inmunológicos/fisiología , Proliferación Celular/efectos de los fármacos , Humanos , Interleucina-10/biosíntesis , Receptor Leucocitario Tipo Inmunoglobulina B1 , Leucocitos Mononucleares/efectos de los fármacos , Leucocitos Mononucleares/patología , Glándula Tiroides/metabolismo
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