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J Med Chem ; 48(23): 7186-91, 2005 Nov 17.
Artículo en Inglés | MEDLINE | ID: mdl-16279776

RESUMEN

Less toxic drugs are needed to combat the human parasite Trypanosoma cruzi (Chagas's disease). One novel target for antitrypanosomal drug design is farnesyltransferase. Several farnesyltransferase inhibitors based on the benzophenone scaffold were assayed in vitro and in vivo with the parasite. The common structural feature of all inhibitors is an amino function which can be protonated. Best in vitro activity (LC50 values 1 and 10 nM, respectively) was recorded for the R-phenylalanine derivative 4a and for the N-propylpiperazinyl derivative 2f. These inhibitors showed no cytotoxicity to cells. When tested in vivo, the survival rates of infected animals receiving the inhibitors at 7 mg/kg body weight/day were 80 and 60% at day 115 postinfection, respectively.


Asunto(s)
Benzofenonas/síntesis química , Farnesiltransferasa/antagonistas & inhibidores , Tripanocidas/síntesis química , Trypanosoma cruzi/efectos de los fármacos , Animales , Benzofenonas/química , Benzofenonas/farmacología , Enfermedad de Chagas/mortalidad , Enfermedad de Chagas/parasitología , Farnesiltransferasa/química , Masculino , Ratones , Ratones Endogámicos BALB C , Modelos Moleculares , Subunidades de Proteína/química , Homología de Secuencia de Aminoácido , Relación Estructura-Actividad , Tripanocidas/química , Tripanocidas/farmacología , Trypanosoma cruzi/enzimología
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